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Quoted phrase not found in phrase index: "Defective DNA repair after ultraviolet radiation damage"
Page 1
Molecular mechanisms of DNA damage recognition for mammalian nucleotide excision repair.
Sugasawa K. Sugasawa K. DNA Repair (Amst). 2016 Aug;44:110-117. doi: 10.1016/j.dnarep.2016.05.015. Epub 2016 May 20. DNA Repair (Amst). 2016. PMID: 27264556 Review.
For faithful DNA repair, it is crucial for cells to locate lesions precisely within the vast genome. ...Unlike XPC, which acts as a versatile sensor of DNA helical distortion, the UV-damaged DNA-binding protein (UV-DDB) is more specialized, reco …
For faithful DNA repair, it is crucial for cells to locate lesions precisely within the vast genome. ...Unlike XPC, which acts …
Human DNA repair disorders in dermatology: A historical perspective, current concepts and new insight.
Moriwaki S. Moriwaki S. J Dermatol Sci. 2016 Feb;81(2):77-84. doi: 10.1016/j.jdermsci.2015.09.008. Epub 2015 Oct 1. J Dermatol Sci. 2016. PMID: 26493104 Review.
To protect against such UV-induced DNA damage, living organisms nicely retain "DNA repair systems", which can efficiently repair "harmful" DNA damage through precise mechanisms by the integrated functions of many proteins. ...In th …
To protect against such UV-induced DNA damage, living organisms nicely retain "DNA repair systems", which can ef …
Cell cycle checkpoints and DNA repair preserve the stability of the human genome.
Kaufmann WK. Kaufmann WK. Cancer Metastasis Rev. 1995 Mar;14(1):31-41. doi: 10.1007/BF00690209. Cancer Metastasis Rev. 1995. PMID: 7606819 Review.
For both initiation of chemical carcinogenesis and UV-clastogenesis, it appears that replication of damaged DNA is required and DNA repair before replication reduces cellular risk. If DNA repair is protective, cell cycle checkpoints which …
For both initiation of chemical carcinogenesis and UV-clastogenesis, it appears that replication of damaged DNA is required an …
Zinc Prevents DNA Damage in Normal Cells but Shows Genotoxic and Cytotoxic Effects in Acute Myeloid Leukemia Cells.
Costa MI, Lapa BS, Jorge J, Alves R, Carreira IM, Sarmento-Ribeiro AB, Gonçalves AC. Costa MI, et al. Int J Mol Sci. 2022 Feb 25;23(5):2567. doi: 10.3390/ijms23052567. Int J Mol Sci. 2022. PMID: 35269710 Free PMC article.
Genomic instability is prevented by the DNA damage response (DDR). Micronutrients, like zinc (Zn), are cofactors of DDR proteins, and micronutrient deficiencies have been related to increased cancer risk. ...In contrast, in normal lymphocytes, supplementation decrea …
Genomic instability is prevented by the DNA damage response (DDR). Micronutrients, like zinc (Zn), are cofactors of DDR protei …
The beneficial role of functional food components in mitigating ultraviolet-induced skin damage.
Morifuji M. Morifuji M. Exp Dermatol. 2019 Feb;28 Suppl 1:28-31. doi: 10.1111/exd.13825. Exp Dermatol. 2019. PMID: 30698876 Review.
Excessive exposure to ultraviolet (UV) radiation can chemically alter biological molecules and is one of the major environmental health risks with potential to damage the structure and function of the skin. ...Milk fermented with lactic acid bacteria has been …
Excessive exposure to ultraviolet (UV) radiation can chemically alter biological molecules and is one of the major environment …
Phosphorylation of xeroderma pigmentosum group C regulates ultraviolet-induced DNA damage repair.
Shah P, Zhao B, Qiang L, He YY. Shah P, et al. Nucleic Acids Res. 2018 Jun 1;46(10):5050-5060. doi: 10.1093/nar/gky239. Nucleic Acids Res. 2018. PMID: 29660033 Free PMC article.
Nucleotide excision repair (NER) is the most versatile DNA repair system that removes bulky DNA damage induced by various endogenous and exogenous factors, including UV radiation. ...Our findings have identified XPC phosphorylation as a n …
Nucleotide excision repair (NER) is the most versatile DNA repair system that removes bulky DNA damage in …
Hydrochlorothiazide Use and Risk of Nonmelanoma Skin Cancers: A Biological Plausibility Study.
Bigagli E, Cinci L, D'Ambrosio M, Nardini P, Portelli F, Colucci R, Lodovici M, Mugelli A, Luceri C. Bigagli E, et al. Oxid Med Cell Longev. 2021 Aug 14;2021:6655542. doi: 10.1155/2021/6655542. eCollection 2021. Oxid Med Cell Longev. 2021. PMID: 34434485 Free PMC article.
Under acute exposure, therapeutically relevant concentrations of HCTZ (70, 140, and 370 ng/mL) amplified UVA-induced double-strand breaks, oxidative DNA, and protein damage in HaCaT human keratinocytes, and this effect was associated to a defective activity o …
Under acute exposure, therapeutically relevant concentrations of HCTZ (70, 140, and 370 ng/mL) amplified UVA-induced double-strand breaks, o …
Repair of UV light-induced DNA damage and risk of cutaneous malignant melanoma.
Wei Q, Lee JE, Gershenwald JE, Ross MI, Mansfield PF, Strom SS, Wang LE, Guo Z, Qiao Y, Amos CI, Spitz MR, Duvic M. Wei Q, et al. J Natl Cancer Inst. 2003 Feb 19;95(4):308-15. doi: 10.1093/jnci/95.4.308. J Natl Cancer Inst. 2003. PMID: 12591987
Patients with xeroderma pigmentosum, a disease characterized by severe sensitivity to UV radiation and a defect in nucleotide excision repair, have a high incidence of CMM, which suggests that DNA repair capacity (DRC) plays a role in sunlight-i …
Patients with xeroderma pigmentosum, a disease characterized by severe sensitivity to UV radiation and a defect in nucleotide …
Ultraviolet-sensitive syndrome cells are defective in transcription-coupled repair of cyclobutane pyrimidine dimers.
Spivak G, Itoh T, Matsunaga T, Nikaido O, Hanawalt P, Yamaizumi M. Spivak G, et al. DNA Repair (Amst). 2002 Aug 6;1(8):629-43. doi: 10.1016/s1568-7864(02)00056-3. DNA Repair (Amst). 2002. PMID: 12509286
Complementation studies with hereditary DNA repair syndromes show that UV(S)S is distinct from all known xeroderma pigmentosum (XP) and Cockayne syndrome (CS) groups. ...Those observations have led us to suggest that UV(S)S cells, like those from CS, are defectiv …
Complementation studies with hereditary DNA repair syndromes show that UV(S)S is distinct from all known xeroderma pigmentosum …
Clinical symptoms and DNA repair characteristics of xeroderma pigmentosum patients from Germany.
Thielmann HW, Popanda O, Edler L, Jung EG. Thielmann HW, et al. Cancer Res. 1991 Jul 1;51(13):3456-70. Cancer Res. 1991. PMID: 2054785
After treatment with UV light or N-acetoxy-2-acetylaminofluorene, cell strains in which DNA-incising capacity was reduced also showed a similar reduction in both colony-forming ability and DNA repair synthesis. ...Weighted means of DNA repair sy …
After treatment with UV light or N-acetoxy-2-acetylaminofluorene, cell strains in which DNA-incising capacity was reduced also showed …
76 results