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Quoted phrase not found in phrase index: "Defective DNA repair after ultraviolet radiation damage"
Page 1
Phosphorylation of xeroderma pigmentosum group C regulates ultraviolet-induced DNA damage repair.
Shah P, Zhao B, Qiang L, He YY. Shah P, et al. Nucleic Acids Res. 2018 Jun 1;46(10):5050-5060. doi: 10.1093/nar/gky239. Nucleic Acids Res. 2018. PMID: 29660033 Free PMC article.
Nucleotide excision repair (NER) is the most versatile DNA repair system that removes bulky DNA damage induced by various endogenous and exogenous factors, including UV radiation. ...Our findings have identified XPC phosphorylation as a n …
Nucleotide excision repair (NER) is the most versatile DNA repair system that removes bulky DNA damage in …
Mutations in UVSSA cause UV-sensitive syndrome and destabilize ERCC6 in transcription-coupled DNA repair.
Zhang X, Horibata K, Saijo M, Ishigami C, Ukai A, Kanno S, Tahara H, Neilan EG, Honma M, Nohmi T, Yasui A, Tanaka K. Zhang X, et al. Nat Genet. 2012 May;44(5):593-7. doi: 10.1038/ng.2228. Nat Genet. 2012. PMID: 22466612
UV-sensitive syndrome (UV(S)S) is an autosomal recessive disorder characterized by photosensitivity and deficiency in transcription-coupled repair (TCR), a subpathway of nucleotide-excision repair that rapidly removes transcription-blocking DNA damage. …
UV-sensitive syndrome (UV(S)S) is an autosomal recessive disorder characterized by photosensitivity and deficiency in transcription-coupled …
Cellular hypersensitivity to UV-A: a clue to the aetiology of actinic reticuloid?
Giannelli F, Botcherby PK, Marimo B, Magnus IA. Giannelli F, et al. Lancet. 1983 Jan 15;1(8316):88-91. doi: 10.1016/s0140-6736(83)91740-3. Lancet. 1983. PMID: 6184584
Fibroblasts cultured from six patients with actinic reticuloid (AR) showed striking cytopathic changes and inhibition of RNA synthesis after exposure to near-ultraviolet radiation that had no effect on normal and other photosensitive cell strains, such as those of x …
Fibroblasts cultured from six patients with actinic reticuloid (AR) showed striking cytopathic changes and inhibition of RNA synthesis after …
Radiation therapy for high-risk squamous cell carcinomas in patients with xeroderma pigmentosum: report of two cases and review of the literature.
Schaffer JV, Orlow SJ. Schaffer JV, et al. Dermatology. 2011;223(2):97-103. doi: 10.1159/000324509. Epub 2011 Oct 21. Dermatology. 2011. PMID: 22024645 Review.
Radiation therapy (RT) represents an important adjuvant treatment modality for high-risk squamous cell carcinomas (SCCs). ...A review of reported cases revealed that XP patients generally have normal cellular and clinical responses to ionizing radiation, which refle …
Radiation therapy (RT) represents an important adjuvant treatment modality for high-risk squamous cell carcinomas (SCCs). ...A review …
The comet assay as a repair test for prenatal diagnosis of Xeroderma pigmentosum and trichothiodystrophy.
Alapetite C, Benoit A, Moustacchi E, Sarasin A. Alapetite C, et al. J Invest Dermatol. 1997 Feb;108(2):154-9. doi: 10.1111/1523-1747.ep12332692. J Invest Dermatol. 1997. PMID: 9008227 Free article.
Xeroderma pigmentosum (XP) and trichothiodystrophy (TTD) are autosomal recessive diseases associated with extreme cutaneous photosensitivity, a defect in nucleotide excision repair (NER), and genetic complexity. ...The comet assay and the UDS test were performed in …
Xeroderma pigmentosum (XP) and trichothiodystrophy (TTD) are autosomal recessive diseases associated with extreme cutaneous photosensitivity …
Enhanced host cell reactivation of a UV-damaged reporter gene in pre-UV-treated cells is delayed in Cockayne syndrome cells.
Pitsikas P, Francis MA, Rainbow AJ. Pitsikas P, et al. J Photochem Photobiol B. 2005 Nov 1;81(2):89-97. doi: 10.1016/j.jphotobiol.2005.06.001. Epub 2005 Aug 25. J Photochem Photobiol B. 2005. PMID: 16125967
We have used a non-replicating recombinant adenovirus, Ad5HCMVlacZ, which expresses the beta-galactosidase (beta-gal) reporter gene, to examine the time course of UV-inducible repair of UV-damaged DNA in human fibroblasts. Host cell reactivation (HCR) …
We have used a non-replicating recombinant adenovirus, Ad5HCMVlacZ, which expresses the beta-galactosidase (beta-gal) reporter gene, to exam …
Deficient expression of enhanced reactivation of parvovirus H-1 in ataxia telangiectasia cells irradiated with X-rays or u.v. light.
Hilgers G, Chen YQ, Cornelis JJ, Rommelaere J. Hilgers G, et al. Carcinogenesis. 1987 Feb;8(2):315-9. doi: 10.1093/carcin/8.2.315. Carcinogenesis. 1987. PMID: 3802417
Cells of patients with ataxia telangiectasia (AT), an inherited disease characterized by a high propensity to cancer, are hypersensitive to ionizing radiation. We investigated whether the hyper-radiosensitivity of AT cells correlated with a defect in their constitut …
Cells of patients with ataxia telangiectasia (AT), an inherited disease characterized by a high propensity to cancer, are hypersensitive to …