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2005 1
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Mannich Base PIP-199 Is a Chemically Unstable Pan-Assay Interference Compound.
Wu X, Krishna Sudhakar H, Alcock LJ, Lau YH. Wu X, et al. J Med Chem. 2023 Aug 24;66(16):11271-11281. doi: 10.1021/acs.jmedchem.3c00674. Epub 2023 Aug 9. J Med Chem. 2023. PMID: 37555818
Mannich base PIP-199 is the only reported small-molecule inhibitor of the Fanconi anemia complementation group M-RecQ-mediated genome instability protein (FANCM-RMI), a protein-protein interaction that governs genome instability in the genetic d …
Mannich base PIP-199 is the only reported small-molecule inhibitor of the Fanconi anemia complementation group …
Diagnostic significance of stromal changes in biopsies of prostate adenocarcinoma.
Dzaparidze G, Kazachonok D, Gvozdkov A, Taelma H, Laht K, Minajeva A. Dzaparidze G, et al. Pathol Res Pract. 2021 Jun;222:153436. doi: 10.1016/j.prp.2021.153436. Epub 2021 Apr 8. Pathol Res Pract. 2021. PMID: 33857855
The research aims to evaluate the potential of stromal changes as a supplementary tool to predict the presence of higher grade carcinomas in the prostate using Masson's trichrome and Fanconi anemia complementation group M (FANCM) antibody staini …
The research aims to evaluate the potential of stromal changes as a supplementary tool to predict the presence of higher grade carcinomas in …
Characterization and acceleration of genome shuffling and ploidy reduction in synthetic allopolyploids by genome sequencing and editing.
Zhang X, Zhang S, Liu Z, Zhao W, Zhang X, Song J, Jia H, Yang W, Ma Y, Wang Y, Xie K, Budahn H, Wang H. Zhang X, et al. Nucleic Acids Res. 2023 Jan 11;51(1):198-217. doi: 10.1093/nar/gkac1209. Nucleic Acids Res. 2023. PMID: 36583364 Free PMC article.
To accelerate genome shuffling, we developed an efficient genome editing platform for Raphanobrassica. By editing Fanconi Anemia Complementation Group M (FANCM) genes, homoeologous recombination, chromosome deletion and secondary meiosis with ad …
To accelerate genome shuffling, we developed an efficient genome editing platform for Raphanobrassica. By editing Fanconi Anemia …
Association of FANCM Mutations with Familial and Early-Onset Breast Cancer Risk in a South American Population.
Morales-Pison S, Morales-González S, Fernandez-Ramires R, Tapia JC, Maldonado E, Calaf GM, Jara L. Morales-Pison S, et al. Int J Mol Sci. 2023 Feb 17;24(4):4041. doi: 10.3390/ijms24044041. Int J Mol Sci. 2023. PMID: 36835452 Free PMC article.
BRCA1/2 are responsible for 16-20% of the risk for hereditary BC. Other susceptibility genes have been identified; Fanconi Anemia Complementation Group M (FANCM) being one of these. ...
BRCA1/2 are responsible for 16-20% of the risk for hereditary BC. Other susceptibility genes have been identified; Fanconi Anemia …
Bi-allelic Recessive Loss-of-Function Variants in FANCM Cause Non-obstructive Azoospermia.
Kasak L, Punab M, Nagirnaja L, Grigorova M, Minajeva A, Lopes AM, Punab AM, Aston KI, Carvalho F, Laasik E, Smith LB; GEMINI Consortium; Conrad DF, Laan M. Kasak L, et al. Am J Hum Genet. 2018 Aug 2;103(2):200-212. doi: 10.1016/j.ajhg.2018.07.005. Am J Hum Genet. 2018. PMID: 30075111 Free PMC article.
Compound heterozygous loss-of-function (LoF) variants in FANCM (Fanconi anemia complementation group M) were detected as the most likely cause for their condition. ...
Compound heterozygous loss-of-function (LoF) variants in FANCM (Fanconi anemia complementation group M) w …
Impaired FANCD2 monoubiquitination and hypersensitivity to camptothecin uniquely characterize Fanconi anemia complementation group M.
Singh TR, Bakker ST, Agarwal S, Jansen M, Grassman E, Godthelp BC, Ali AM, Du CH, Rooimans MA, Fan Q, Wahengbam K, Steltenpool J, Andreassen PR, Williams DA, Joenje H, de Winter JP, Meetei AR. Singh TR, et al. Blood. 2009 Jul 2;114(1):174-80. doi: 10.1182/blood-2009-02-207811. Epub 2009 May 7. Blood. 2009. PMID: 19423727 Free PMC article.
The Fanconi anemia ortholog FANCM ensures ordered homologous recombination in both somatic and meiotic cells in Arabidopsis.
Knoll A, Higgins JD, Seeliger K, Reha SJ, Dangel NJ, Bauknecht M, Schröpfer S, Franklin FC, Puchta H. Knoll A, et al. Plant Cell. 2012 Apr;24(4):1448-64. doi: 10.1105/tpc.112.096644. Epub 2012 Apr 30. Plant Cell. 2012. PMID: 22547783 Free PMC article.
It is caused by single mutations in the FANC genes, one of which encodes the DNA translocase FANCM (for Fanconi anemia complementation group M), which is required for the repair of DNA interstrand cross-links to ensure replication progression. . …
It is caused by single mutations in the FANC genes, one of which encodes the DNA translocase FANCM (for Fanconi anemia comp …
Improved Genome Editing through Inhibition of FANCM and Members of the BTR Dissolvase Complex.
de Alencastro G, Puzzo F, Pavel-Dinu M, Zhang F, Pillay S, Majzoub K, Tiffany M, Jang H, Sheikali A, Cromer MK, Meetei R, Carette JE, Porteus MH, Pekrun K, Kay MA. de Alencastro G, et al. Mol Ther. 2021 Mar 3;29(3):1016-1027. doi: 10.1016/j.ymthe.2020.10.020. Epub 2020 Oct 22. Mol Ther. 2021. PMID: 33678249 Free PMC article.
In order to improve our understanding of the mechanism behind TI mediated by AAV and improve its efficiency, we performed an unbiased genetic screen in human cells using a promoterless AAV-homologous recombination (AAV-HR) vector system. We identified that the inhibition of the …
In order to improve our understanding of the mechanism behind TI mediated by AAV and improve its efficiency, we performed an unbiased geneti …
Prevalence of FANCM germline variants in BRCA1/2 negative breast and/or ovarian cancer patients from Pakistan.
Rashid MU, Muhammad N, Shehzad U, Khan FA, Loya A, Hamann U. Rashid MU, et al. Fam Cancer. 2023 Jan;22(1):31-41. doi: 10.1007/s10689-022-00304-1. Epub 2022 Jul 8. Fam Cancer. 2023. PMID: 35802266
The Fanconi anemia complementation group M (FANCM) gene is a potential candidate for breast/ovarian cancer susceptibility in European populations. ...
The Fanconi anemia complementation group M (FANCM) gene is a potential candidate for breast/ovarian cance …
12 results