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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1985 2
1991 1
1992 1
1994 1
1995 2
1996 2
1997 5
1999 3
2000 5
2001 3
2002 2
2003 4
2004 2
2005 2
2006 4
2007 4
2008 3
2009 1
2010 2
2012 2
2015 2
2016 1
2018 1
2020 1
2021 2
2024 0

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56 results

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Page 1
Effects of Low-Carbohydrate versus Mediterranean Diets on Weight Loss, Glucose Metabolism, Insulin Kinetics and β-Cell Function in Morbidly Obese Individuals.
Tricò D, Moriconi D, Berta R, Baldi S, Quinones-Galvan A, Guiducci L, Taddei S, Mari A, Nannipieri M. Tricò D, et al. Nutrients. 2021 Apr 18;13(4):1345. doi: 10.3390/nu13041345. Nutrients. 2021. PMID: 33919503 Free PMC article. Clinical Trial.
The two dietary regimens proved similarly effective in improving insulin resistance and fasting hyperinsulinemia, while enhancing endogenous insulin clearance and beta-cell glucose sensitivity. ...
The two dietary regimens proved similarly effective in improving insulin resistance and fasting hyperinsulinemia, while enhanc …
Attenuation of age- and sucrose-induced insulin resistance and syndrome X by a synergistic antioxidant cocktail: the AMIS syndrome and HISS hypothesis.
Lautt WW, Ming Z, Legare DJ. Lautt WW, et al. Can J Physiol Pharmacol. 2010 Mar;88(3):313-23. doi: 10.1139/Y09-130. Can J Physiol Pharmacol. 2010. PMID: 20393596 Free PMC article. Review.
Rats given the sucrose supplement had accelerated AMIS and developed fasting hyperinsulinemia and postprandial hyperglycemia, hyperlipidemia, hyperinsulinemia, and adiposity. ...
Rats given the sucrose supplement had accelerated AMIS and developed fasting hyperinsulinemia and postprandial hyperglycemia, …
Inhibition of insulin secretion as a new drug target in the treatment of metabolic disorders.
Hansen JB, Arkhammar PO, Bodvarsdottir TB, Wahl P. Hansen JB, et al. Curr Med Chem. 2004 Jun;11(12):1595-615. doi: 10.2174/0929867043365026. Curr Med Chem. 2004. PMID: 15180566 Review.
Deficient insulin release causes hyperglycemia and diabetes, whereas excessive insulin release can give rise to serious metabolic disorders, such as nesidioblastosis (Persistent Hyperinsulinemic Hypoglycemia of Infancy, PHHI) and might also be closely associated with development …
Deficient insulin release causes hyperglycemia and diabetes, whereas excessive insulin release can give rise to serious metabolic disorders, …
Metformin therapy decreases hyperandrogenism and hyperinsulinemia in women with polycystic ovary syndrome.
Kolodziejczyk B, Duleba AJ, Spaczynski RZ, Pawelczyk L. Kolodziejczyk B, et al. Fertil Steril. 2000 Jun;73(6):1149-54. doi: 10.1016/s0015-0282(00)00501-x. Fertil Steril. 2000. PMID: 10856473 Free article. Clinical Trial.
OBJECTIVE: To evaluate the effects of 12 weeks of metformin therapy on hormonal and clinical indices in polycystic ovary syndrome (PCOS). DESIGN: Prospective study. SETTING: University hospital. PATIENT(S): Thirty-nine women with PCOS and fasting hyperinsulinemia
OBJECTIVE: To evaluate the effects of 12 weeks of metformin therapy on hormonal and clinical indices in polycystic ovary syndrome (PC …
Fungal lysozyme leverages the gut microbiota to curb DSS-induced colitis.
Larsen IS, Jensen BAH, Bonazzi E, Choi BSY, Kristensen NN, Schmidt EGW, Süenderhauf A, Morin L, Olsen PB, Hansen LBS, Schröder T, Sina C, Chassaing B, Marette A. Larsen IS, et al. Gut Microbes. 2021 Jan-Dec;13(1):1988836. doi: 10.1080/19490976.2021.1988836. Gut Microbes. 2021. PMID: 34693864 Free PMC article.
We assessed longitudinal microbiome alterations by 16S amplicon sequencing in both models.Lysozyme dose-dependently alleviated intestinal inflammation in DSS-challenged mice and further protected against HFD-induced microbiota encroachment and fasting hyperinsulinemia
We assessed longitudinal microbiome alterations by 16S amplicon sequencing in both models.Lysozyme dose-dependently alleviated intestinal in …
Fasting hyperinsulinemia in human immunodeficiency virus-infected men: relationship to body composition, gonadal function, and protease inhibitor use.
Hadigan C, Corcoran C, Stanley T, Piecuch S, Klibanski A, Grinspoon S. Hadigan C, et al. J Clin Endocrinol Metab. 2000 Jan;85(1):35-41. doi: 10.1210/jcem.85.1.6264. J Clin Endocrinol Metab. 2000. PMID: 10634360 Clinical Trial.
Insulin responses to testosterone administration were investigated among 52 HIV-infected men with hypogonadism and wasting (weight <90% ideal body weight and/or weight loss >10%) randomized to either testosterone (300 mg, im, every 3 weeks) or placebo for 6 months. . …
Insulin responses to testosterone administration were investigated among 52 HIV-infected men with hypogonadism and wasting (weight <90% i …
Distribution and cutoff points of fasting insulin in Asian Indian adolescents and their association with metabolic syndrome.
Vikram NK, Misra A, Pandey RM, Luthra K, Bhatt SP. Vikram NK, et al. J Assoc Physicians India. 2008 Dec;56:949-54. J Assoc Physicians India. 2008. PMID: 19322974
The derived cutoff points for fasting insulin (pmol/L) were: 14-15 y- 128.5 and 164.8; 16-17 y- 126.1 and 152.8; 18-19 y- 121.2 and 162.4 in males and females, respectively. Prevalence of fasting hyperinsulinemia (39.1%) and MS (NCEP 2.2%, IDF 1.5%) was highest in a …
The derived cutoff points for fasting insulin (pmol/L) were: 14-15 y- 128.5 and 164.8; 16-17 y- 126.1 and 152.8; 18-19 y- 121.2 and 162.4 in …
Insulin resistance syndrome in children : pathophysiology and potential management strategies.
Decsi T, Molnár D. Decsi T, et al. Paediatr Drugs. 2003;5(5):291-9. doi: 10.2165/00128072-200305050-00002. Paediatr Drugs. 2003. PMID: 12716216 Review.
The clustering of risk factors can be termed insulin resistance syndrome (IRS) because of the putative central role of tissue insulin insensitivity in the background of the inter-related metabolic disturbances. Fasting hyperinsulinemia, impaired glucose tolerance, d …
The clustering of risk factors can be termed insulin resistance syndrome (IRS) because of the putative central role of tissue insulin insens …
56 results