Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1967 1
1968 1
1969 2
1970 1
1971 2
1972 1
1973 1
1974 1
1975 4
1976 1
1977 6
1978 3
1979 6
1980 6
1981 4
1982 1
1983 6
1984 8
1985 3
1986 4
1987 8
1988 5
1989 13
1990 7
1991 5
1992 7
1993 4
1994 7
1995 18
1996 6
1997 3
1998 8
1999 11
2000 9
2001 9
2002 10
2003 15
2004 18
2005 19
2006 21
2007 25
2008 25
2009 22
2010 28
2011 21
2012 24
2013 34
2014 50
2015 32
2016 40
2017 45
2018 32
2019 40
2020 54
2021 44
2022 35
2023 40
2024 14

Text availability

Article attribute

Article type

Publication date

Search Results

799 results

Results by year

Filters applied: . Clear all
Page 1
G6PD deficiency: An update.
Harcke SJ, Rizzolo D, Harcke HT. Harcke SJ, et al. JAAPA. 2019 Nov;32(11):21-26. doi: 10.1097/01.JAA.0000586304.65429.a7. JAAPA. 2019. PMID: 31609781
Although glucose-6-phosphate dehydrogenase (G6PD) deficiency is less known in Western countries than in the Middle East and Africa, global migration and immigration are bringing ethnic groups with the highest incidence of this inherited genetic disorder into the US …
Although glucose-6-phosphate dehydrogenase (G6PD) deficiency is less known in Western countries than in the Middle East and Af …
Favism: Clinical Features at Different Ages.
Beretta A, Manuelli M, Cena H. Beretta A, et al. Nutrients. 2023 Jan 10;15(2):343. doi: 10.3390/nu15020343. Nutrients. 2023. PMID: 36678214 Free PMC article. Review.
Favism is a hemolytic disease due to the ingestion of fava beans in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency. There is wide inter- and intra-individual variability in the development of hemolytic crisis, and several factors influence it: …
Favism is a hemolytic disease due to the ingestion of fava beans in individuals with glucose-6-phosphate dehydrogenase (G6PD) defi …
Medications and glucose-6-phosphate dehydrogenase deficiency: an evidence-based review.
Youngster I, Arcavi L, Schechmaster R, Akayzen Y, Popliski H, Shimonov J, Beig S, Berkovitch M. Youngster I, et al. Drug Saf. 2010 Sep 1;33(9):713-26. doi: 10.2165/11536520-000000000-00000. Drug Saf. 2010. PMID: 20701405 Review.
Although drug-induced haemolysis is considered the most common adverse clinical consequence of G6PD deficiency, significant confusion exists regarding which drugs can cause haemolytic anaemia in patients with G6PD deficiency. ...After assessing the lit …
Although drug-induced haemolysis is considered the most common adverse clinical consequence of G6PD deficiency, significant co …
Tafenoquine and G6PD: a primer for clinicians.
Chu CS, Freedman DO. Chu CS, et al. J Travel Med. 2019 Jun 1;26(4):taz023. doi: 10.1093/jtm/taz023. J Travel Med. 2019. PMID: 30941413 Free PMC article. Review.
As with other 8-aminoquinolines, tafenoquine causes hemolysis in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency (hemizygous males and homozygous females) and is contraindicated in this population. ...Testing for G6PD deficiency is …
As with other 8-aminoquinolines, tafenoquine causes hemolysis in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficie …
Association between G6PD deficiency and hyperbilirubinemia in neonates: a meta-analysis.
Liu H, Liu W, Tang X, Wang T. Liu H, et al. Pediatr Hematol Oncol. 2015 Mar;32(2):92-8. doi: 10.3109/08880018.2014.887803. Epub 2014 Mar 31. Pediatr Hematol Oncol. 2015. PMID: 24684295 Review.
However, many pediatricians did not take into account the probable effect of G6PD-deficiency when severe neonatal hyperbilirubinemia occurred. ...This study revealed a significant correlation between G6PD-deficiency and neonatal hyperbilirubinemia, as …
However, many pediatricians did not take into account the probable effect of G6PD-deficiency when severe neonatal hyperbilirub …
Incorporating G6PD genotyping to identify patients with G6PD deficiency.
Morris SA, Crews KR, Hayden RT, Takemoto CM, Yang W, Baker DK, Broeckel U, Relling MV, Haidar CE. Morris SA, et al. Pharmacogenet Genomics. 2022 Apr 1;32(3):87-93. doi: 10.1097/FPC.0000000000000456. Pharmacogenet Genomics. 2022. PMID: 34693927 Free PMC article.
Glucose-6-phosphate-dehydrogenase (G6PD) deficiency is a common X-linked enzyme disorder associated with hemolytic anemia after exposure to fava beans or certain medications. Activity testing is the gold standard for detecting G6PD deficiency; however, …
Glucose-6-phosphate-dehydrogenase (G6PD) deficiency is a common X-linked enzyme disorder associated with hemolytic anemia afte …
Glucose-6-phosphate dehydrogenase deficiency.
Mehta A, Mason PJ, Vulliamy TJ. Mehta A, et al. Baillieres Best Pract Res Clin Haematol. 2000 Mar;13(1):21-38. doi: 10.1053/beha.1999.0055. Baillieres Best Pract Res Clin Haematol. 2000. PMID: 10916676 Review.
Glucose-6-phosphate dehydrogenase (G6PD) is expressed in all tissues, where it catalyses the first step in the pentose phosphate pathway. G6PD deficiency is prevalent throughout tropical and subtropical regions of the world because of the protection it affords durin …
Glucose-6-phosphate dehydrogenase (G6PD) is expressed in all tissues, where it catalyses the first step in the pentose phosphate pathway. …
Component therapy.
Radhakrishnan KM, Chakravarthi S, Pushkala S, Jayaraju J. Radhakrishnan KM, et al. Indian J Pediatr. 2003 Aug;70(8):661-6. doi: 10.1007/BF02724257. Indian J Pediatr. 2003. PMID: 14510088 Review.
The advantage of fresh cells is that hyperkalemia is avoided and good post transfusion survival acceptable red cell oxygen affinity. However it has to be screened for sickle cell disease and G6PD deficiency. Indications for exchange transfusion are kernicterus, neon …
The advantage of fresh cells is that hyperkalemia is avoided and good post transfusion survival acceptable red cell oxygen affinity. However …
Non-nutritional anemia: Malaria, thalassemia, G6PD deficiency and tuberculosis in Indonesia.
Malik SG, Oktavianthi S, Wahlqvist ML, Asih PBS, Harahap A, Satyagraha AW, Syafruddin D. Malik SG, et al. Asia Pac J Clin Nutr. 2020;29(Suppl 1):S32-S40. doi: 10.6133/apjcn.202012_29(S1).04. Asia Pac J Clin Nutr. 2020. PMID: 33377745 Free article. Review.
This review focusses on two interrelated nonnutritional causes of anemia: malaria infection and RBC disorders (thalassemia and G6PD deficiency), as well as tuberculosis. According to the Haldane hypothesis, thalassemia occurs as a protective trait toward malaria inf …
This review focusses on two interrelated nonnutritional causes of anemia: malaria infection and RBC disorders (thalassemia and G6PD …
G6PD diaxBox: Digital image-based quantification of G6PD deficiency.
Palasuwan D, Chalidabhongse TH, Chancharoen R, Palasuwan A, Kobchaisawat T, Phanomchoeng G. Palasuwan D, et al. Talanta. 2021 Oct 1;233:122538. doi: 10.1016/j.talanta.2021.122538. Epub 2021 May 26. Talanta. 2021. PMID: 34215041 Free article.
Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzymopathy in humans. More than 400 million people worldwide are affected by this genetic condition. Testing for G6PD deficiency before drug administration is essential for patient …
Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzymopathy in humans. More than 400 million people wor …
799 results