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Quoted phrase not found in phrase index: "Glioma susceptibility 1"
Page 1
Genome-wide association study of glioma subtypes identifies specific differences in genetic susceptibility to glioblastoma and non-glioblastoma tumors.
Melin BS, Barnholtz-Sloan JS, Wrensch MR, Johansen C, Il'yasova D, Kinnersley B, Ostrom QT, Labreche K, Chen Y, Armstrong G, Liu Y, Eckel-Passow JE, Decker PA, Labussière M, Idbaih A, Hoang-Xuan K, Di Stefano AL, Mokhtari K, Delattre JY, Broderick P, Galan P, Gousias K, Schramm J, Schoemaker MJ, Fleming SJ, Herms S, Heilmann S, Nöthen MM, Wichmann HE, Schreiber S, Swerdlow A, Lathrop M, Simon M, Sanson M, Andersson U, Rajaraman P, Chanock S, Linet M, Wang Z, Yeager M; GliomaScan Consortium; Wiencke JK, Hansen H, McCoy L, Rice T, Kosel ML, Sicotte H, Amos CI, Bernstein JL, Davis F, Lachance D, Lau C, Merrell RT, Shildkraut J, Ali-Osman F, Sadetzki S, Scheurer M, Shete S, Lai RK, Claus EB, Olson SH, Jenkins RB, Houlston RS, Bondy ML. Melin BS, et al. Nat Genet. 2017 May;49(5):789-794. doi: 10.1038/ng.3823. Epub 2017 Mar 27. Nat Genet. 2017. PMID: 28346443 Free PMC article.
Genome-wide association studies (GWAS) have transformed our understanding of glioma susceptibility, but individual studies have had limited power to identify risk loci. ...We identified five new loci for glioblastoma (GBM) at 1p31.3 (rs12752552; P = 2.04 10(-9), odd …
Genome-wide association studies (GWAS) have transformed our understanding of glioma susceptibility, but individual studies hav …
Melanoma genetics.
Read J, Wadt KA, Hayward NK. Read J, et al. J Med Genet. 2016 Jan;53(1):1-14. doi: 10.1136/jmedgenet-2015-103150. Epub 2015 Sep 3. J Med Genet. 2016. PMID: 26337759 Review.
Although the majority of genetic alterations associated with melanoma development are somatic, the underlying presence of heritable melanoma risk genes is an important component of disease occurrence. Susceptibility for some families is due to mutation in one of the …
Although the majority of genetic alterations associated with melanoma development are somatic, the underlying presence of heritable melanoma …
Gliomas in Children.
Filbin MG, Sturm D. Filbin MG, et al. Semin Neurol. 2018 Feb;38(1):121-130. doi: 10.1055/s-0038-1635106. Epub 2018 Mar 16. Semin Neurol. 2018. PMID: 29548059 Review.
Although the exact cells of origin for most pediatric gliomas remain to be identified, our current understanding is that specific cell populations during CNS development are susceptible to particular oncogenic events during certain time windows and thus give rise to …
Although the exact cells of origin for most pediatric gliomas remain to be identified, our current understanding is that specific cel …
Origin of Gliomas.
Cahill D, Turcan S. Cahill D, et al. Semin Neurol. 2018 Feb;38(1):5-10. doi: 10.1055/s-0037-1620238. Epub 2018 Mar 16. Semin Neurol. 2018. PMID: 29548046 Review.
Malignant glioma is a common type of brain tumor that remains largely incurable. ...Herein we summarize our current knowledge on the population epidemiology of gliomas, heritable genetic risk factors, the somatic events that contribute to tumor evolution, and mouse …
Malignant glioma is a common type of brain tumor that remains largely incurable. ...Herein we summarize our current knowledge on the …
Telomere maintenance and the etiology of adult glioma.
Walsh KM, Wiencke JK, Lachance DH, Wiemels JL, Molinaro AM, Eckel-Passow JE, Jenkins RB, Wrensch MR. Walsh KM, et al. Neuro Oncol. 2015 Nov;17(11):1445-52. doi: 10.1093/neuonc/nov082. Epub 2015 May 25. Neuro Oncol. 2015. PMID: 26014050 Free PMC article. Review.
Common inherited variants near the telomerase-component genes TERC and TERT are associated both with longer telomere length and increased risk of glioma. Exome sequencing of glioma patients from families with multiple affected members has identified rare inherited m …
Common inherited variants near the telomerase-component genes TERC and TERT are associated both with longer telomere length and increased ri …
XPG gene polymorphisms and glioma susceptibility: a two-centre case-control study.
Yuan L, Hu WM, Chen K, Shi Q, Lin A, Chen HT, Zhuo ZJ, Zeng L. Yuan L, et al. Br J Biomed Sci. 2021 Jul;78(3):135-140. doi: 10.1080/09674845.2020.1870308. Epub 2021 Feb 5. Br J Biomed Sci. 2021. PMID: 33393424
Xeroderma pigmentosum group G (XPG) plays a key role in the nucleotide excision repair pathway, which may modulate individual susceptibility to developing cancer. We hypothesized links between XPG variants and glioma in children.Methods: We tested our hypothesis in …
Xeroderma pigmentosum group G (XPG) plays a key role in the nucleotide excision repair pathway, which may modulate individual susceptibil …
Role of PVT1 polymorphisms in the glioma susceptibility and prognosis.
Ding X, Zhao Y, Yuan H, Zhang Y, Gao Y. Ding X, et al. Eur J Cancer Prev. 2021 Sep 1;30(5):400-408. doi: 10.1097/CEJ.0000000000000636. Eur J Cancer Prev. 2021. PMID: 33443959
BACKGROUND: Genetic factors play a crucial role in the glioma risk and prognosis of glioma patients. To explore the role of plasmacytoma variant translocation 1 (PVT1) polymorphism in the susceptibility and survival of glioma in the Chinese Han …
BACKGROUND: Genetic factors play a crucial role in the glioma risk and prognosis of glioma patients. To explore the role of pl …
Epidemiological characteristics and genetic alterations in adult diffuse glioma in East Asian populations.
Mo Z, Xin J, Chai R, Woo PYM, Chan DTM, Wang J. Mo Z, et al. Cancer Biol Med. 2022 Nov 1;19(10):1440-59. doi: 10.20892/j.issn.2095-3941.2022.0418. Cancer Biol Med. 2022. PMID: 36350002 Free PMC article. Review.
For instance, risk loci 12p11.23, 15q15-21.1, and 19p13.12 were reported in East Asians, whereas risk loci 8q24.21, 1p31.3, and 1q32.1 were reported in studies in White patients. ...By summarizing large-scale disease surveillance, germline, and somatic genomi …
For instance, risk loci 12p11.23, 15q15-21.1, and 19p13.12 were reported in East Asians, whereas risk loci 8q24.21, 1p31.3, and 1q32. …
Association Between LIN28A Gene Polymorphisms and Glioma Susceptibility in Chinese Children.
Guo H, Liao Y, Lin A, Lin H, Huang X, Ruan J, Yuan L, Zhuo Z. Guo H, et al. Cancer Control. 2021 Jan-Dec;28:10732748211040009. doi: 10.1177/10732748211040009. Cancer Control. 2021. PMID: 34468231 Free PMC article.
The occurrence and development of various malignant tumors is closely related with LIN28A gene, but its relationship with glioma susceptibility has not been widely discovered. In this case-control study, we conducted four single nucleotide polymorphisms (SNPs) (rs38 …
The occurrence and development of various malignant tumors is closely related with LIN28A gene, but its relationship with glioma s …
Testing for causality between systematically identified risk factors and glioma: a Mendelian randomization study.
Howell AE, Robinson JW, Wootton RE, McAleenan A, Tsavachidis S, Ostrom QT, Bondy M, Armstrong G, Relton C, Haycock P, Martin RM, Zheng J, Kurian KM. Howell AE, et al. BMC Cancer. 2020 Jun 3;20(1):508. doi: 10.1186/s12885-020-06967-2. BMC Cancer. 2020. PMID: 32493226 Free PMC article.
Using MR, we found evidence that four genetically predicted traits increased risk of glioma, glioblastoma or non-glioblastoma: longer leukocyte telomere length, liability to allergic disease, increased alcohol consumption and liability to childhood extreme obesity ( …
Using MR, we found evidence that four genetically predicted traits increased risk of glioma, glioblastoma or non-glioblastoma: longer …
772 results