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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1971 1
1982 1
1989 1
1991 1
1993 1
1994 1
1996 2
1997 1
1999 2
2000 2
2001 1
2002 2
2003 3
2004 2
2005 1
2006 2
2008 3
2009 3
2010 3
2011 5
2012 4
2013 3
2014 8
2015 8
2016 5
2017 6
2018 6
2019 5
2020 7
2021 4
2022 4
2023 4
2024 1

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85 results

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Page 1
Pathophysiology of Glucocorticoid Signaling.
Vitellius G, Trabado S, Bouligand J, Delemer B, Lombès M. Vitellius G, et al. Ann Endocrinol (Paris). 2018 Jun;79(3):98-106. doi: 10.1016/j.ando.2018.03.001. Epub 2018 Apr 20. Ann Endocrinol (Paris). 2018. PMID: 29685454 Review.
Adrenocorticotropin resistance syndromes.
Cooray SN, Chan L, Metherell L, Storr H, Clark AJL. Cooray SN, et al. Endocr Dev. 2008;13:99-116. doi: 10.1159/000134828. Endocr Dev. 2008. PMID: 18493136 Review.
Familial glucocorticoid deficiency (FGD) and triple A syndrome belong to a rare group of autosomal recessive disorders characterized by adrenocorticotropin (ACTH) insensitivity. Unlike triple A syndrome which presents a range of clinical features, FGD is solely char …
Familial glucocorticoid deficiency (FGD) and triple A syndrome belong to a rare group of autosomal recessive disorders charact …
ACTH resistance syndromes.
Huebner A, Elias LL, Clark AJ. Huebner A, et al. J Pediatr Endocrinol Metab. 1999 Apr;12 Suppl 1:277-93. J Pediatr Endocrinol Metab. 1999. PMID: 10698592 Review.
This review focuses on two autosomal recessive disorders, familial glucocorticoid deficiency (FGD) (MIM*202200) and the triple A syndrome (MIM*231550), which have at least three different molecular aetiologies. ...
This review focuses on two autosomal recessive disorders, familial glucocorticoid deficiency (FGD) (MIM*202200) and the triple …
Optimal glucocorticoid replacement in adrenal insufficiency.
Øksnes M, Ross R, Løvås K. Øksnes M, et al. Best Pract Res Clin Endocrinol Metab. 2015 Jan;29(1):3-15. doi: 10.1016/j.beem.2014.09.009. Epub 2014 Oct 7. Best Pract Res Clin Endocrinol Metab. 2015. PMID: 25617168 Review.
Adrenal insufficiency (glucocorticoid deficiency) comprises a group of rare diseases, including primary adrenal insufficiency, secondary adrenal insufficiency and congenital adrenal hyperplasia. ...
Adrenal insufficiency (glucocorticoid deficiency) comprises a group of rare diseases, including primary adrenal insufficiency, …
Substitution therapy in adult patients with congenital adrenal hyperplasia.
Reisch N. Reisch N. Best Pract Res Clin Endocrinol Metab. 2015 Jan;29(1):33-45. doi: 10.1016/j.beem.2014.11.002. Epub 2014 Nov 14. Best Pract Res Clin Endocrinol Metab. 2015. PMID: 25617171 Review.
Steroid 21-hydroxylase deficiency (21OHD) is its most prevalent form, accounting for over 90% of all cases. Clinically classic 21OHD is characterised by glucocorticoid deficiency and adrenal androgen excess with (salt wasting form) or without (simple virilising form …
Steroid 21-hydroxylase deficiency (21OHD) is its most prevalent form, accounting for over 90% of all cases. Clinically classic 21OHD is char …
GDF15 Is Elevated in Conditions of Glucocorticoid Deficiency and Is Modulated by Glucocorticoid Replacement.
Melvin A, Chantzichristos D, Kyle CJ, Mackenzie SD, Walker BR, Johannsson G, Stimson RH, O'Rahilly S. Melvin A, et al. J Clin Endocrinol Metab. 2020 May 1;105(5):1427-34. doi: 10.1210/clinem/dgz277. J Clin Endocrinol Metab. 2020. PMID: 31853550 Free PMC article. Clinical Trial.
Anorexia, weight loss, nausea and vomiting are common manifestations of glucocorticoid deficiency, and we hypothesized that glucocorticoid deficiency may be associated with elevated levels of GDF15. ...Given the site of action of GDF15 in the hindbrain …
Anorexia, weight loss, nausea and vomiting are common manifestations of glucocorticoid deficiency, and we hypothesized that …
Hyponatremia and antidiuresis syndrome.
Vantyghem MC, Balavoine AS, Wémeau JL, Douillard C. Vantyghem MC, et al. Ann Endocrinol (Paris). 2011 Dec;72(6):500-12. doi: 10.1016/j.ando.2011.10.001. Epub 2011 Nov 25. Ann Endocrinol (Paris). 2011. PMID: 22119069 Review.
Salt loss of gastrointestinal, renal, cutaneous and sometimes cerebral origin is hypovolaemic, hypoosmolar hyponatraemia (skin tenting), whereas oedema is present with hypervolaemic, hypoosmolar hyponatraemia of heart failure, nephrotic syndrome and cirrhosis. Some endocrinopathi …
Salt loss of gastrointestinal, renal, cutaneous and sometimes cerebral origin is hypovolaemic, hypoosmolar hyponatraemia (skin tenting), whe …
85 results