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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1957 1
1960 1
1964 1
1969 1
1970 1
1974 1
1975 4
1976 7
1977 7
1978 6
1979 10
1980 6
1981 5
1982 9
1983 14
1984 10
1985 16
1986 10
1987 12
1988 14
1989 18
1990 19
1991 21
1992 18
1993 27
1994 13
1995 23
1996 23
1997 22
1998 38
1999 46
2000 33
2001 37
2002 46
2003 41
2004 38
2005 38
2006 40
2007 61
2008 42
2009 51
2010 41
2011 45
2012 55
2013 57
2014 64
2015 61
2016 62
2017 65
2018 61
2019 60
2020 55
2021 44
2022 47
2023 28
2024 10

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1,434 results

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Quoted phrase not found in phrase index: "Holoprosencephaly 12 with or without pancreatic agenesis"
Page 1
Identification and management of pancreas divisum.
Gutta A, Fogel E, Sherman S. Gutta A, et al. Expert Rev Gastroenterol Hepatol. 2019 Nov;13(11):1089-1105. doi: 10.1080/17474124.2019.1685871. Epub 2019 Nov 8. Expert Rev Gastroenterol Hepatol. 2019. PMID: 31663403 Free PMC article. Review.
In our attempt to offer clarity, a comprehensive search on PubMed, Ovid, Embase and Cochrane Library from inception to May 2019 was undertaken using key words "pancreas divisum", "idiopathic recurrent acute pancreatitis" and "chronic pancreatitis".Expert opin …
In our attempt to offer clarity, a comprehensive search on PubMed, Ovid, Embase and Cochrane Library from inception to May 2019 was undertak …
Inter-organ crosstalk during development and progression of type 2 diabetes mellitus.
Xourafa G, Korbmacher M, Roden M. Xourafa G, et al. Nat Rev Endocrinol. 2024 Jan;20(1):27-49. doi: 10.1038/s41574-023-00898-1. Epub 2023 Oct 16. Nat Rev Endocrinol. 2024. PMID: 37845351 Review.
Type 2 diabetes mellitus (T2DM) is characterized by tissue-specific insulin resistance and pancreatic beta-cell dysfunction, which result from the interplay of local abnormalities within different tissues and systemic dysregulation of tissue crosstalk. ...Starting f …
Type 2 diabetes mellitus (T2DM) is characterized by tissue-specific insulin resistance and pancreatic beta-cell dysfunction, which re …
Pancreas divisum.
DiMagno MJ, Wamsteker EJ. DiMagno MJ, et al. Curr Gastroenterol Rep. 2011 Apr;13(2):150-6. doi: 10.1007/s11894-010-0170-8. Curr Gastroenterol Rep. 2011. PMID: 21222060 Free PMC article. Review.
We review important new clinical observations in pancreas divisum (PD) made since 2007. PD is common and has the same prevalence in the general population and idiopathic pancreatitis (IP). Up to 53% of patients with PD and IP have underlying idiopathic chroni …
We review important new clinical observations in pancreas divisum (PD) made since 2007. PD is common and has the same prevalen …
Postburn pancreatitis.
Ryan CM, Sheridan RL, Schoenfeld DA, Warshaw AL, Tompkins RG. Ryan CM, et al. Ann Surg. 1995 Aug;222(2):163-70. doi: 10.1097/00000658-199508000-00008. Ann Surg. 1995. PMID: 7543741 Free PMC article.
OBJECTIVE: The authors examined the prevalence and complications of pancreatitis in severely burned patients. Factors predictive for the development of pancreatitis after burns are considered. ...Most of these patients (40/49, 82%) had symptoms of pancreat …
OBJECTIVE: The authors examined the prevalence and complications of pancreatitis in severely burned patients. Factors predictive …
Acute pancreatitis.
Geokas MC, Baltaxe HA, Banks PA, Silva J Jr, Frey CF. Geokas MC, et al. Ann Intern Med. 1985 Jul;103(1):86-100. doi: 10.7326/0003-4819-103-1-86. Ann Intern Med. 1985. PMID: 3890660 Review.
In-vivo autodigestion is prevented by an array of natural safeguards. In acute pancreatitis, inappropriate intrapancreatic activation and release of pancreatic hydrolases occur, but the pathogenetic mechanism of autodigestion is unclear. The release of proteases, li …
In-vivo autodigestion is prevented by an array of natural safeguards. In acute pancreatitis, inappropriate intrapancreatic activation …
Isoniazid-induced recurrent pancreatitis.
Mattioni S, Zamy M, Mechai F, Raynaud JJ, Chabrol A, Aflalo V, Biour M, Bouchaud O. Mattioni S, et al. JOP. 2012 May 10;13(3):314-6. JOP. 2012. PMID: 22572141 Free article. Review.
Eight days later, the same treatment was reintroduced and the patient presented recurrent pancreatitis; thus, treatment was withheld again followed by disappearance of clinical and biological abnormalities. Two days later, a treatment without isoniazid was reintrodu …
Eight days later, the same treatment was reintroduced and the patient presented recurrent pancreatitis; thus, treatment was withheld …
Exocrine Pancreas Dysfunction in Type 1 Diabetes.
Foster TP, Bruggeman B, Campbell-Thompson M, Atkinson MA, Haller MJ, Schatz DA. Foster TP, et al. Endocr Pract. 2020 Dec;26(12):1505-1513. doi: 10.4158/EP-2020-0295. Endocr Pract. 2020. PMID: 33471743 Free PMC article. Review.
OBJECTIVE: Type 1 diabetes (T1D) is characterized by autoimmune beta-cell destruction, but exocrine pancreas abnormalities may also play a role in the disease pathophysiology. ...T cells, dendritic cells, neutrophils, and products of complement activation are seen i …
OBJECTIVE: Type 1 diabetes (T1D) is characterized by autoimmune beta-cell destruction, but exocrine pancreas abnormalities may …
Structure and function of the exocrine pancreas in patients with type 1 diabetes.
Alexandre-Heymann L, Mallone R, Boitard C, Scharfmann R, Larger E. Alexandre-Heymann L, et al. Rev Endocr Metab Disord. 2019 Jun;20(2):129-149. doi: 10.1007/s11154-019-09501-3. Rev Endocr Metab Disord. 2019. PMID: 31077020 Review.
This arose the question of the relationships between the endocrine and exocrine parts of the pancreas in T1D pathogenesis. Our review underlines that histological anomalies of the exocrine pancreas are common in patients with T1D: intralobular and interacinar fibros …
This arose the question of the relationships between the endocrine and exocrine parts of the pancreas in T1D pathogenesis. Our review …
"Biliary Diseases with Pancreatic Counterparts": Cross-sectional Imaging Findings.
Katabathina VS, Flaherty EM, Dasyam AK, Menias CO, Riddle ND, Lath N, Kozaka K, Matsui O, Nakanuma Y, Prasad SR. Katabathina VS, et al. Radiographics. 2016 Mar-Apr;36(2):374-92. doi: 10.1148/rg.2016150071. Epub 2016 Feb 1. Radiographics. 2016. PMID: 26824512 Review.
Both nonneoplastic and neoplastic pathologic conditions of the biliary tract have their counterparts in the pancreas. Immunoglobulin G4 (IgG4)-related sclerosing cholangitis is the biliary manifestation of IgG4-related sclerosing disease, and type 1 autoimmune pancreati …
Both nonneoplastic and neoplastic pathologic conditions of the biliary tract have their counterparts in the pancreas. Immunoglobulin …
Pancreas divisum.
Varshney S, Johnson CD. Varshney S, et al. Int J Pancreatol. 1999 Apr;25(2):135-41. doi: 10.1385/IJGC:25:2:135. Int J Pancreatol. 1999. PMID: 10360226 Review.
Clinically, symptomatic patients with PD may be divided into five groups: group 1, those with minimal symptoms; group 2, those with recurrent acute pancreatitis or upper abdominal pain with no other cause; group 3, those with chronic pancreatitis; group 4, those wit …
Clinically, symptomatic patients with PD may be divided into five groups: group 1, those with minimal symptoms; group 2, those with recurren …
1,434 results