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113 results

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Quoted phrase not found in phrase index: "Hypoalphalipoproteinemia, primary, 1"
Page 1
Cholesterol Efflux Pathways Suppress Inflammasome Activation, NETosis, and Atherogenesis.
Westerterp M, Fotakis P, Ouimet M, Bochem AE, Zhang H, Molusky MM, Wang W, Abramowicz S, la Bastide-van Gemert S, Wang N, Welch CL, Reilly MP, Stroes ES, Moore KJ, Tall AR. Westerterp M, et al. Circulation. 2018 Aug 28;138(9):898-912. doi: 10.1161/CIRCULATIONAHA.117.032636. Circulation. 2018. PMID: 29588315 Free PMC article.
Nlrp3 or Caspase-1/11 deficiency decreased atherosclerotic lesion size in myeloid Abca1/g1-deficient Ldlr(-/-) mice. Myeloid Abca1/g1 deficiency enhanced caspase-1 cleavage not only in splenic monocytes and macrophages, but also in neutrophils, and dramatically enha …
Nlrp3 or Caspase-1/11 deficiency decreased atherosclerotic lesion size in myeloid Abca1/g1-deficient Ldlr(-/-) mice. Myeloid Abca1/g1 …
ABCA1 and atherosclerosis.
Soumian S, Albrecht C, Davies AH, Gibbs RG. Soumian S, et al. Vasc Med. 2005 May;10(2):109-19. doi: 10.1191/1358863x05vm593ra. Vasc Med. 2005. PMID: 16013195 Review.
Primary and familial hypoalphalipoproteinemia.
Third JL, Montag J, Flynn M, Freidel J, Laskarzewski P, Glueck CJ. Third JL, et al. Metabolism. 1984 Feb;33(2):136-46. doi: 10.1016/0026-0495(84)90126-4. Metabolism. 1984. PMID: 6694557
Our specific aim was to assess within-family clustering of high-density lipoprotein cholesterol (HDLC) levels in kindreds identified through probands with primary hypoalphalipoproteinemia, and to determine whether, and to what degree, familial aggregation of HDLC le …
Our specific aim was to assess within-family clustering of high-density lipoprotein cholesterol (HDLC) levels in kindreds identified through …
Lipoprotein-X fifty years after its original discovery.
Fellin R, Manzato E. Fellin R, et al. Nutr Metab Cardiovasc Dis. 2019 Jan;29(1):4-8. doi: 10.1016/j.numecd.2018.09.006. Epub 2018 Sep 26. Nutr Metab Cardiovasc Dis. 2019. PMID: 30503707 Review.
The frequency of cardiovascular events in patients affected by primary biliary cholangitis, in whom the Lp-X is present in high concentration, are not increased. ...
The frequency of cardiovascular events in patients affected by primary biliary cholangitis, in whom the Lp-X is present in high conce …
High-density lipoprotein subpopulations in pathologic conditions.
Asztalos BF, Schaefer EJ. Asztalos BF, et al. Am J Cardiol. 2003 Apr 3;91(7A):12E-17E. doi: 10.1016/s0002-9149(02)03383-0. Am J Cardiol. 2003. PMID: 12679198 Review.
Our studies of apo A-I-containing HDL subpopulations in various patient populations reveal that patients homozygous for Tangier disease have only the pre-beta(1) HDL subspecies. Tangier heterozygotes are severely depleted in the larger alpha- and pre-alpha-mobility subspec …
Our studies of apo A-I-containing HDL subpopulations in various patient populations reveal that patients homozygous for Tangier disease have …
Diagnosis and treatment of high density lipoprotein deficiency.
Schaefer EJ, Anthanont P, Diffenderfer MR, Polisecki E, Asztalos BF. Schaefer EJ, et al. Prog Cardiovasc Dis. 2016 Sep-Oct;59(2):97-106. doi: 10.1016/j.pcad.2016.08.006. Epub 2016 Aug 24. Prog Cardiovasc Dis. 2016. PMID: 27565770 Free PMC article. Review.
Patients with marked deficiency of HDL-C (<20 mg/dL) in the absence of secondary causes are much less common (<1% of the population). These patients may have homozygous, compound heterozygous, or heterozygous defects involving the apolipoprotein (APO)AI, ABCA1, or le …
Patients with marked deficiency of HDL-C (<20 mg/dL) in the absence of secondary causes are much less common (<1% of the popula …
Genetic and secondary causes of severe HDL deficiency and cardiovascular disease.
Geller AS, Polisecki EY, Diffenderfer MR, Asztalos BF, Karathanasis SK, Hegele RA, Schaefer EJ. Geller AS, et al. J Lipid Res. 2018 Dec;59(12):2421-2435. doi: 10.1194/jlr.M088203. Epub 2018 Oct 17. J Lipid Res. 2018. PMID: 30333156 Free PMC article.
Mutations (23 novel) and selected variants were found at the following gene loci: 1) ABCA1 (26.9%): 2 homozygotes, 7 compound or double heterozygotes, 30 heterozygotes, and 2 homozygotes and 13 heterozygotes with variants rs9282541/p.R230C or rs111292742/c.-279C>G; 2) L …
Mutations (23 novel) and selected variants were found at the following gene loci: 1) ABCA1 (26.9%): 2 homozygotes, 7 compound or doub …
Update on dyslipidemia.
Garg A, Simha V. Garg A, et al. J Clin Endocrinol Metab. 2007 May;92(5):1581-9. doi: 10.1210/jc.2007-0275. J Clin Endocrinol Metab. 2007. PMID: 17483372 Review.
Linkage of familial combined hyperlipidemia to upstream stimulatory factor 1 remains controversial. Recent guidelines of the Adult Treatment Panel III emphasize intensive reduction of LDL or non-high-density lipoprotein cholesterol in patients at high risk of CHD. ...
Linkage of familial combined hyperlipidemia to upstream stimulatory factor 1 remains controversial. Recent guidelines of the Adult Tr …
Analysis of familial hypoalphalipoproteinemia syndromes.
Frohlich J, Pritchard PH. Frohlich J, et al. Mol Cell Biochem. 1992 Aug 18;113(2):141-9. doi: 10.1007/BF00231534. Mol Cell Biochem. 1992. PMID: 1518505 Review.
Familial hypoalphalipoproteinemias (HA) are a heterogenous group of disorders characterized by various degrees of HDL deficiency. Differential diagnosis involves clinical and biochemical evaluation after intervention designed to correct known secondary causes of low HDL. T …
Familial hypoalphalipoproteinemias (HA) are a heterogenous group of disorders characterized by various degrees of HDL deficiency. Dif …
113 results