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Page 1
Malignant McLeod myopathy.
Jung HH, Brandner S. Jung HH, et al. Muscle Nerve. 2002 Sep;26(3):424-7. doi: 10.1002/mus.10199. Muscle Nerve. 2002. PMID: 12210375
Other possible causes for rhabdomyolysis such as prolonged immobility, trauma, hyperthermia, generalized seizures, toxin exposure, or metabolic changes were excluded. Clinical course was favorable, with persistent slight elevation of serum creatine kinase levels caused by …
Other possible causes for rhabdomyolysis such as prolonged immobility, trauma, hyperthermia, generalized seizures, toxin exposure, or metabo …
Chronic granulomatous disease and McLeod syndrome: Stem cell transplant and transfusion support in a 2-year-old patient-a case report.
Helander L, McKinney C, Kelly K, Mack S, Sanders M, Gurley J, Dumont LJ, Annen K. Helander L, et al. Front Immunol. 2022 Aug 23;13:994321. doi: 10.3389/fimmu.2022.994321. eCollection 2022. Front Immunol. 2022. PMID: 36081507 Free PMC article.
Reduced-intensity conditioning was used to reduce the transplant-related mortality risk associated with myeloablative protocols. The transplant course was uneventful; autologous red blood cell (RBC) transfusion support was successful and allowed for the avoidance of possib …
Reduced-intensity conditioning was used to reduce the transplant-related mortality risk associated with myeloablative protocols. The transpl …
McLeod myopathy revisited: more neurogenic and less benign.
Hewer E, Danek A, Schoser BG, Miranda M, Reichard R, Castiglioni C, Oechsner M, Goebel HH, Heppner FL, Jung HH. Hewer E, et al. Brain. 2007 Dec;130(Pt 12):3285-96. doi: 10.1093/brain/awm269. Brain. 2007. PMID: 18055495 Free article.
McLeod phenotype associated with a XK missense mutation without hematologic, neuromuscular, or cerebral involvement.
Jung HH, Hergersberg M, Vogt M, Pahnke J, Treyer V, Röthlisberger B, Kollias SS, Russo D, Frey BM. Jung HH, et al. Transfusion. 2003 Jul;43(7):928-38. doi: 10.1046/j.1537-2995.2003.t01-1-00434.x. Transfusion. 2003. PMID: 12823753
CONCLUSION: Known disease-causing XK gene mutations comprised deletions, nonsense, or splice-site mutations predicting absent or truncated XK protein devoid of the Kell-protein binding site. ...
CONCLUSION: Known disease-causing XK gene mutations comprised deletions, nonsense, or splice-site mutations predicting absent or trun …