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Quoted phrase not found in phrase index: "Red cell phospholipid defect with hemolysis"
Page 1
Mechanism of erythrocyte death in human population exposed to arsenic through drinking water.
Biswas D, Banerjee M, Sen G, Das JK, Banerjee A, Sau TJ, Pandit S, Giri AK, Biswas T. Biswas D, et al. Toxicol Appl Pharmacol. 2008 Jul 1;230(1):57-66. doi: 10.1016/j.taap.2008.02.003. Epub 2008 Feb 15. Toxicol Appl Pharmacol. 2008. PMID: 18377941 Clinical Trial.
Arsenic toxicity increased membrane microviscosity along with an elevation of cholesterol/phospholipid ratio, which hampered the flexibility of red cell membrane and made them less deformable. ...Arsenic toxicity intervened into red cell membran …
Arsenic toxicity increased membrane microviscosity along with an elevation of cholesterol/phospholipid ratio, which hampered the flex …
Correction of the PNH defect by GPI-anchored protein transfer.
Sloand EM, Maciejewski JP, Dunn D, Moss J, Brewer B, Kirby M, Young NS. Sloand EM, et al. Blood. 1998 Dec 1;92(11):4439-45. Blood. 1998. PMID: 9834251 Free article.
Both in vivo and in vitro models have suggested the feasibility of cell-to-cell transfer of GPI proteins, and patients with hemolysis could potentially benefit from transfer of CD59 to their deficient erythrocytes. ...PNH RBC that were exposed to HDL, …
Both in vivo and in vitro models have suggested the feasibility of cell-to-cell transfer of GPI proteins, and patients with …
Intrinsic defects in erythroid cells from familial hemophagocytic lymphohistiocytosis type 5 patients identify a role for STXBP2/Munc18-2 in erythropoiesis and phospholipid scrambling.
Kostova EB, Beuger BM, Veldthuis M, van der Werff Ten Bosch J, Kühnle I, van den Akker E, van den Berg TK, van Zwieten R, van Bruggen R. Kostova EB, et al. Exp Hematol. 2015 Dec;43(12):1072-1076.e2. doi: 10.1016/j.exphem.2015.08.007. Epub 2015 Aug 28. Exp Hematol. 2015. PMID: 26320718 Free article. Clinical Trial.
Here we report that red blood cells express Munc18-2 and that erythroid cells from patients with FHL-5 exhibit intrinsic defects caused by STXBP2/Munc18-2 mutations. ...Furthermore, cultured erythroblasts from patients with FHL-5 display defective erythropoie …
Here we report that red blood cells express Munc18-2 and that erythroid cells from patients with FHL-5 exhibit intrinsic defects
Temporal sequence of major biochemical events during blood bank storage of packed red blood cells.
Karon BS, van Buskirk CM, Jaben EA, Hoyer JD, Thomas DD. Karon BS, et al. Blood Transfus. 2012 Oct;10(4):453-61. doi: 10.2450/2012.0099-11. Epub 2012 Mar 28. Blood Transfus. 2012. PMID: 22507860 Free PMC article. Clinical Trial.
BACKGROUND: We used sensitive spectroscopic techniques to measure changes in Band 3 oligomeric state during storage of packed red blood cells (RBC); these changes were compared to metabolic changes, RBC morphology, cholesterol and membrane protein loss, phospholipid
BACKGROUND: We used sensitive spectroscopic techniques to measure changes in Band 3 oligomeric state during storage of packed red blo …
Eculizumab treatment modifies the immune profile of PNH patients.
Alfinito F, Ruggiero G, Sica M, Udhayachandran A, Rubino V, Della Pepa R, Palatucci AT, Annunziatella M, Notaro R, Risitano AM, Terrazzano G. Alfinito F, et al. Immunobiology. 2012 Jul;217(7):698-703. doi: 10.1016/j.imbio.2011.11.009. Epub 2011 Dec 3. Immunobiology. 2012. PMID: 22206707
Numerous data suggest a role for immune-mediated mechanisms in the selection/expansion of GPI-defective clone. Haemolytic anaemia in PNH is dependent on the effect of complement against GPI-defective red cells. ...No effects were observed on NK and Treg. The …
Numerous data suggest a role for immune-mediated mechanisms in the selection/expansion of GPI-defective clone. Haemolytic anaemia in …