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Quoted phrase not found in phrase index: "Reduced CD95-induced lymphocyte apoptosis"
Page 1
Residual CD95-pathway function in children with autoimmune lymphoproliferative syndrome is independent from clinical state and genotype of CD95 mutation.
Fuchs H, Posovszky C, Lahr G, van der Werff ten Bosch J, Boehler T, Debatin KM. Fuchs H, et al. Pediatr Res. 2009 Feb;65(2):163-8. doi: 10.1203/PDR.0b013e318191f7e4. Pediatr Res. 2009. PMID: 18948840
Human autoimmune lymphoproliferative syndrome (ALPS) is caused by defective CD95-mediated apoptosis of lymphocytes. In most patients, heterozygous mutations within the CD95 gene are found. ...All CD95-mutations led to a reduced DISC formation and diminished i …
Human autoimmune lymphoproliferative syndrome (ALPS) is caused by defective CD95-mediated apoptosis of lymphocytes. In most pa …
SHIP-1 inhibits CD95/APO-1/Fas-induced apoptosis in primary T lymphocytes and T leukemic cells by promoting CD95 glycosylation independently of its phosphatase activity.
Charlier E, Condé C, Zhang J, Deneubourg L, Di Valentin E, Rahmouni S, Chariot A, Agostinis P, Pang PC, Haslam SM, Dell A, Penninger J, Erneux C, Piette J, Gloire G. Charlier E, et al. Leukemia. 2010 Apr;24(4):821-32. doi: 10.1038/leu.2010.9. Epub 2010 Feb 11. Leukemia. 2010. PMID: 20147977 Free article.
Using primary T cells from SHIP-1(-/-) mice and T leukemic cell lines, we report that SHIP-1 is a potent inhibitor of CD95-induced death. We observed that a small fraction of the SHIP-1 pool is localized to the endoplasmic reticulum (ER), in which it promotes …
Using primary T cells from SHIP-1(-/-) mice and T leukemic cell lines, we report that SHIP-1 is a potent inhibitor of CD95-induced …
Thiopental-induced apoptosis in lymphocytes is independent of CD95 activation.
Keel M, Mica L, Stover J, Stocker R, Trentz O, Härter L. Keel M, et al. Anesthesiology. 2005 Sep;103(3):576-84. doi: 10.1097/00000542-200509000-00021. Anesthesiology. 2005. PMID: 16129983 Free article.
In contrast, anti-CD95-induced apoptosis in Jurkat cells (27.0 +/- 2.0%) was completely blocked by z-VAD-fmk (8.1 +/- 1.8%). ...The nuclear factor kappaB inhibitor gliotoxin accelerated both thiopental- and CD95-mediated apoptosis, indicating a mutual …
In contrast, anti-CD95-induced apoptosis in Jurkat cells (27.0 +/- 2.0%) was completely blocked by z-VAD-fmk (8.1 +/- 1 …
Inhibition of TNF-alpha mediated cell death by HIV-1 specific protease inhibitors.
Wolf T, Findhammer S, Nolte B, Helm EB, Brodt HR. Wolf T, et al. Eur J Med Res. 2003 Jan 28;8(1):17-24. Eur J Med Res. 2003. PMID: 12578750
Modulation of apoptosis induced in U937 or Jurkat cells by CD95 (Fas-ligand) or TNF-alpha was measured using flow cytometry using the 7-AAD and annexin/propidium iodide methods. - HIV-1 protease inhibitors reduced TNF-alpha mediated cell death in a dose-dependent ma …
Modulation of apoptosis induced in U937 or Jurkat cells by CD95 (Fas-ligand) or TNF-alpha was measured using flow cytometry using the …
Ceramides that mediate apoptosis reduce glucose uptake and transporter affinity for glucose in human leukaemic cell lines but not in neutrophils.
Ahmed N, Berridge MV. Ahmed N, et al. Pharmacol Toxicol. 2000 Mar;86(3):114-21. doi: 10.1034/j.1600-0773.2000.d01-21.x. Pharmacol Toxicol. 2000. PMID: 10752668
We have demonstrated that CD95-induced apoptosis in a human leukaemic T-cell line resulted in loss of glucose transporter function (Berridge et al. 1996). ...With Jurkat and U937 cells, the inhibitory effects of ceramides on glucose transport were associated …
We have demonstrated that CD95-induced apoptosis in a human leukaemic T-cell line resulted in loss of glucose transport …
In vitro effects of interferon-gamma and tumor necrosis factor-alpha on CD34+ bone marrow progenitor cells from aplastic anemia patients and normal donors.
Welsh JP, Rutherford TR, Flynn J, Foukaneli T, Gordon-Smith EC, Gibson FM. Welsh JP, et al. Hematol J. 2004;5(1):39-46. doi: 10.1038/sj.thj.6200340. Hematol J. 2004. PMID: 14745429
This was to determine whether aplastic CD34+ cells are more sensitive than normal cells to IFN-gamma/TNF-alpha-mediated effects, and whether cytokine-induced CD95 expression can explain the high levels of apoptosis observed in CD34+ cells from aplastic patients. CD3 …
This was to determine whether aplastic CD34+ cells are more sensitive than normal cells to IFN-gamma/TNF-alpha-mediated effects, and whether …
CD226 expression deficiency causes high sensitivity to apoptosis in NK T cells from patients with systemic lupus erythematosus.
Tao D, Shangwu L, Qun W, Yan L, Wei J, Junyan L, Feili G, Boquan J, Jinquan T. Tao D, et al. J Immunol. 2005 Feb 1;174(3):1281-90. doi: 10.4049/jimmunol.174.3.1281. J Immunol. 2005. Retraction in: J Immunol. 2012 Jun 1;188(11):5800. doi: 10.4049/jimmunol.1290020. PMID: 15661884 Retracted.
In the present study we report that NK T cells from active SLE patients are highly sensitive to anti-CD95-induced apoptosis compared with those from normal subjects and inactive SLE patients. ...We therefore conclude that deficient expression of CD226 and sur …
In the present study we report that NK T cells from active SLE patients are highly sensitive to anti-CD95-induced apoptosis …