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Elevated morbidity and mortality in patients with chronic idiopathic hypophosphatemia: a nationwide cohort study.
Kim KJ, Song JE, Kim JH, Hong N, Kim SG, Lee J, Rhee Y. Kim KJ, et al. Front Endocrinol (Lausanne). 2023 Aug 10;14:1229750. doi: 10.3389/fendo.2023.1229750. eCollection 2023. Front Endocrinol (Lausanne). 2023. PMID: 37635983 Free PMC article.
After excluding secondary conditions that could change serum phosphorus levels, we identified 154 patients (76 men and 78 women) with non-secondary and non-renal hypophosphatemia. These hypophosphatemic patients were compared at a ratio of 1:10 with age-, sex-, and …
After excluding secondary conditions that could change serum phosphorus levels, we identified 154 patients (76 men and 78 women) with non-se …
Reflections on TRP and TP/GFR in the definition of renal phosphate loss: conceptual review.
García-Nieto VM, González-Rodríguez JD, Cabrera-Sevilla JE, Martín-Fernández de Basoa MC, Luis-Yanes MI. García-Nieto VM, et al. Pediatr Nephrol. 2023 Nov;38(11):3845-3848. doi: 10.1007/s00467-023-05941-x. Epub 2023 Apr 13. Pediatr Nephrol. 2023. PMID: 37052691 Review.
CONCLUSIONS: TRP and TP/GFR are similar parameters. TRP is more effective than TP/GFR given that in renal hypophosphatemia it is always below 95% and above 95% in reduced phosphatemia and normal kidney proximal tubular function. ...
CONCLUSIONS: TRP and TP/GFR are similar parameters. TRP is more effective than TP/GFR given that in renal hypophosphatemia it …
Yield of diagnostic tests in unexplained renal hypophosphatemia: a case series.
Bech AP, Hoorn EJ, Zietse R, Wetzels JFM, Nijenhuis T. Bech AP, et al. BMC Nephrol. 2018 Sep 4;19(1):220. doi: 10.1186/s12882-018-1017-z. BMC Nephrol. 2018. PMID: 30180816 Free PMC article.
BACKGROUND: Isolated renal hypophosphatemia may be inherited or acquired. An increasing number of patients with unexplained renal hypophosphatemia is being referred to our clinics, but the optimal diagnostic work-up is not known. ...CONCLUSIONS: Oral c …
BACKGROUND: Isolated renal hypophosphatemia may be inherited or acquired. An increasing number of patients with unexplained …
Chronic metabolic acidosis increases the serum concentration of 1,25-dihydroxyvitamin D in humans by stimulating its production rate. Critical role of acidosis-induced renal hypophosphatemia.
Krapf R, Vetsch R, Vetsch W, Hulter HN. Krapf R, et al. J Clin Invest. 1992 Dec;90(6):2456-63. doi: 10.1172/JCI116137. J Clin Invest. 1992. PMID: 1469097 Free PMC article.

Plasma [HCO3-] decreased by 4.5 +/- 0.4 mmol/liter in the low dose and by 9.1 +/- 0.3 mmol/liter (P < 0.001) in the high dose group. Metabolic acidosis induced renal hypophosphatemia, which strongly correlated with the severity of acidosis (Plasma [PO4] on plasma

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Plasma [HCO3-] decreased by 4.5 +/- 0.4 mmol/liter in the low dose and by 9.1 +/- 0.3 mmol/liter (P < 0.001) in the high dose group. Meta

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Rickets then and now.
Harrrison HE, Harrison HC. Harrrison HE, et al. J Pediatr. 1975 Dec;87(6 Pt 2):1144-51. doi: 10.1016/s0022-3476(75)80129-6. J Pediatr. 1975. PMID: 1102648 Review.
These disorders can now be classified more exactly into two groups: those in which there is a deficiency of the active metabolite of vitamin D, 1,25-dihydroxyvitamin D, and those in which there is an abnormality of renal tubular function resulting in renal hypophosphate …
These disorders can now be classified more exactly into two groups: those in which there is a deficiency of the active metabolite of vitamin …
Genetic profile of a large Spanish cohort with hypercalcemia.
García-Castaño A, Madariaga L, Gómez-Conde S, González P, Grau G, Rica I, de Nanclares GP, De la Hoz AB, Aguayo A, Martínez R, Urrutia I, Gaztambide S; Calcium Phosphorus Metabolism Molecular Biology Group; Castaño L. García-Castaño A, et al. Front Endocrinol (Lausanne). 2024 Mar 22;15:1297614. doi: 10.3389/fendo.2024.1297614. eCollection 2024. Front Endocrinol (Lausanne). 2024. PMID: 38586466 Free PMC article.
However, we also found variants in genes associated with primary hyperparathyroidism (GCM2), renal hypophosphatemia with or without rickets (SLC34A1, SLC34A3, SLC9A3R1, VDR, and CYP27B1), DiGeorge syndrome (TBX1 and NEBL), and hypophosphatasia (ALPL). ...
However, we also found variants in genes associated with primary hyperparathyroidism (GCM2), renal hypophosphatemia with or wi …