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Does abnormal neuronal excitability exist in myotonic dystrophy? I. Effects of the antiarrhythmic drug hydroquinidine on slow saccadic eye movements.
Di Costanzo A, Mottola A, Toriello A, Di Iorio G, Tedeschi G, Bonavita V. Di Costanzo A, et al. Neurol Sci. 2000 Apr;21(2):73-80. doi: 10.1007/s100720070099. Neurol Sci. 2000. PMID: 10938184 Clinical Trial.
To gain new insights into the pathophysiology of MD, we determined whether the antiarrhythmic drug hydroquinidine would ameliorate slow saccadic eye movements (SEMs), apathy and hypersomnia in MD patients. SEMs were selected as simple modality for psyc …
To gain new insights into the pathophysiology of MD, we determined whether the antiarrhythmic drug hydroquinidine would ameliorate slow …
Does abnormal neuronal excitability exist in myotonic dystrophy? II. Effects of the antiarrhythmic drug hydroquinidine on apathy and hypersomnia.
Di Costanzo A, Mottola A, Toriello A, Di Iorio G, Tedeschi G, Bonavita V. Di Costanzo A, et al. Neurol Sci. 2000 Apr;21(2):81-6. doi: 10.1007/s100720070100. Neurol Sci. 2000. PMID: 10938185 Clinical Trial.
To gain new insights into the pathophysiology of MD, we determined whether the antiarrhythmic drug hydroquinidine could ameliorate apathy and hypersomnia besides slow saccadic eye movements in these patients. The study was conducted in a randomized …
To gain new insights into the pathophysiology of MD, we determined whether the antiarrhythmic drug hydroquinidine could ameliorate apathy an …