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1986 2
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Page 1
Mutagenicity Profile Induced by UVB Light in Human Xeroderma Pigmentosum Group C Cells.
Quintero-Ruiz N, Corradi C, Moreno NC, de Souza TA, Pereira Castro L, Rocha CRR, Menck CFM. Quintero-Ruiz N, et al. Photochem Photobiol. 2022 May;98(3):713-731. doi: 10.1111/php.13516. Epub 2021 Sep 30. Photochem Photobiol. 2022. PMID: 34516658
The mutation spectra induced by UVB were investigated by whole-exome sequencing of randomly selected clones of NER-proficient and XP-C-deficient human skin fibroblasts. ...
The mutation spectra induced by UVB were investigated by whole-exome sequencing of randomly selected clones of NER-proficient and XP- …
Xeroderma Pigmentosum Group C Deficiency Alters Cigarette Smoke DNA Damage Cell Fate and Accelerates Emphysema Development.
Sears CR, Zhou H, Justice MJ, Fisher AJ, Saliba J, Lamb I, Wicker J, Schweitzer KS, Petrache I. Sears CR, et al. Am J Respir Cell Mol Biol. 2018 Mar;58(3):402-411. doi: 10.1165/rcmb.2017-0251OC. Am J Respir Cell Mol Biol. 2018. PMID: 29111769 Free PMC article.
Using complementary cell culture and mouse models of CS exposure, we investigated the role of the DNA repair protein, xeroderma pigmentosum group C (XPC), on CS-induced DNA damage repair and emphysema. ...
Using complementary cell culture and mouse models of CS exposure, we investigated the role of the DNA repair protein, xeroderma pi …
DNA repair as an emerging target for COPD-lung cancer overlap.
Sears CR. Sears CR. Respir Investig. 2019 Mar;57(2):111-121. doi: 10.1016/j.resinv.2018.11.005. Epub 2019 Jan 7. Respir Investig. 2019. PMID: 30630751 Free article. Review.
We discuss emerging data supporting a key role for the DNA repair protein, xeroderma pigmentosum group C (XPC), in cigarette smoke-induced COPD and early lung cancer development. ...
We discuss emerging data supporting a key role for the DNA repair protein, xeroderma pigmentosum group C (XPC), …
Astragalus polysaccharide protects formaldehyde-induced toxicity by promoting NER pathway in bone marrow mesenchymal stem cells.
She Y, Zhao X, Wu P, Xue L, Liu Z, Zhu M, Yang J, Li Y. She Y, et al. Folia Histochem Cytobiol. 2021;59(2):124-133. doi: 10.5603/FHC.a2021.0013. Epub 2021 May 19. Folia Histochem Cytobiol. 2021. PMID: 34008856 Free article.
MATERIAL AND METHODS: Human BMSCs were cultured in vitro and randomly divided into control cells (Ctrl group), FA-treated cells (FA group, 120 mumol/L), and cells incubated with FA and increasing concentrations (40, 100, or 400 mug/mL) of APS (FA + APS groups). ...The mRNA …
MATERIAL AND METHODS: Human BMSCs were cultured in vitro and randomly divided into control cells (Ctrl group), FA-treated cells (FA g …
A novel missense variant and multiexon deletion causing a delayed presentation of xeroderma pigmentosum, group C.
Macke EL, Morales-Rosado JA, Gupta A, Schmitz CT, Kruisselbrink T, Lanpher B, Klee EW. Macke EL, et al. Cold Spring Harb Mol Case Stud. 2020 Aug 25;6(4):a005165. doi: 10.1101/mcs.a005165. Print 2020 Aug. Cold Spring Harb Mol Case Stud. 2020. PMID: 32843428 Free PMC article.
Pathogenic variants in the XPC complex subunit, DNA damage recognition, and repair factor (XPC) are the cause of xeroderma pigmentosum, group C (MIM: 278720). Xeroderma pigmentosum is an inherited condition characterized by hypersensitivity to ultravio …
Pathogenic variants in the XPC complex subunit, DNA damage recognition, and repair factor (XPC) are the cause of xeroderma pigment …
Excision repair in xeroderma pigmentosum group C but not group D is clustered in a small fraction of the total genome.
Karentz D, Cleaver JE. Karentz D, et al. Mutat Res. 1986 May;165(3):165-74. doi: 10.1016/0167-8817(86)90051-9. Mutat Res. 1986. PMID: 3084965
The repair sites in group C cells were, however, clustered together in small regions of the genome which appeared to be mended nearly as efficiently as the whole genome is mended in normal cells, while repair in group D cells was randomly distributed. Growth of normal cell …
The repair sites in group C cells were, however, clustered together in small regions of the genome which appeared to be mended nearly as eff …
XPC protects against smoking- and carcinogen-induced lung adenocarcinoma.
Zhou H, Saliba J, Sandusky GE, Sears CR. Zhou H, et al. Carcinogenesis. 2019 May 14;40(3):403-411. doi: 10.1093/carcin/bgz003. Carcinogenesis. 2019. PMID: 30624620 Free PMC article.
Cigarette smoke (CS) contains hundreds of carcinogens and is a potent inducer of oxidative and bulky DNA damage, which when insufficiently repaired leads to activation of DNA damage response and possibly mutations. The DNA repair protein xeroderma pigmentosum gro …
Cigarette smoke (CS) contains hundreds of carcinogens and is a potent inducer of oxidative and bulky DNA damage, which when insufficiently r …
Excision repair in xeroderma pigmentosum group C cells is regulated differently in transformed cells and primary fibroblasts.
Cleaver JE. Cleaver JE. Biochem Biophys Res Commun. 1988 Oct 14;156(1):557-62. doi: 10.1016/s0006-291x(88)80878-7. Biochem Biophys Res Commun. 1988. PMID: 2845984
Excision repair in xeroderma pigmentosum group C cells occurs at about 20-30% of normal levels. ...In SV40-transformed fibroblasts and Epstein-Barr virus-transformed lymphocytes of complementation group C, however, excision repair is randomly di …
Excision repair in xeroderma pigmentosum group C cells occurs at about 20-30% of normal levels. ...In SV40-trans …
XPC Lys939Gln polymorphism, smoking and risk of sporadic colorectal cancer among Malaysians.
Ahmad Aizat AA, Siti Nurfatimah MS, Aminudin MM, Ankathil R. Ahmad Aizat AA, et al. World J Gastroenterol. 2013 Jun 21;19(23):3623-8. doi: 10.3748/wjg.v19.i23.3623. World J Gastroenterol. 2013. PMID: 23801864 Free PMC article.
AIM: To investigate the risk association of xeroderma pigmentosum group C (XPC) Lys939Gln polymorphism alone and in combination with cigarette smoking on colorectal cancer (CRC) predisposition. ...
AIM: To investigate the risk association of xeroderma pigmentosum group C (XPC) Lys939Gln polymorphism alone and …
26 results