Aoki Y - Search Results

1

Application of Urine-Derived Stem Cells to Cellular Modeling in Neuromuscular and Neurodegenerative Diseases.

Sato M, Takizawa H, Nakamura A, Turner BJ, Shabanpoor F, Aoki Y. Sato M, et al. Among authors: aoki y. Front Mol Neurosci. 2019 Dec 5;12:297. doi: 10.3389/fnmol.2019.00297. eCollection 2019. Front Mol Neurosci. 2019. PMID: 31920531 Free PMC article. Review.

2

Antisense PMO found in dystrophic dog model was effective in cells from exon 7-deleted DMD patient.

Saito T, Nakamura A, Aoki Y, Yokota T, Okada T, Osawa M, Takeda S. Saito T, et al. Among authors: aoki y. PLoS One. 2010 Aug 18;5(8):e12239. doi: 10.1371/journal.pone.0012239. PLoS One. 2010. PMID: 20805873 Free PMC article.

3

In-frame dystrophin following exon 51-skipping improves muscle pathology and function in the exon 52-deficient mdx mouse.

Aoki Y, Nakamura A, Yokota T, Saito T, Okazawa H, Nagata T, Takeda S. Aoki Y, et al. Mol Ther. 2010 Nov;18(11):1995-2005. doi: 10.1038/mt.2010.186. Epub 2010 Sep 7. Mol Ther. 2010. PMID: 20823833 Free PMC article.

4

Identification of muscle-specific microRNAs in serum of muscular dystrophy animal models: promising novel blood-based markers for muscular dystrophy.

Mizuno H, Nakamura A, Aoki Y, Ito N, Kishi S, Yamamoto K, Sekiguchi M, Takeda S, Hashido K. Mizuno H, et al. Among authors: aoki y. PLoS One. 2011 Mar 30;6(3):e18388. doi: 10.1371/journal.pone.0018388. PLoS One. 2011. PMID: 21479190 Free PMC article.

5

Bodywide skipping of exons 45-55 in dystrophic mdx52 mice by systemic antisense delivery.

Aoki Y, Yokota T, Nagata T, Nakamura A, Tanihata J, Saito T, Duguez SM, Nagaraju K, Hoffman EP, Partridge T, Takeda S. Aoki Y, et al. Proc Natl Acad Sci U S A. 2012 Aug 21;109(34):13763-8. doi: 10.1073/pnas.1204638109. Epub 2012 Aug 6. Proc Natl Acad Sci U S A. 2012. PMID: 22869723 Free PMC article.

6

Highly efficient in vivo delivery of PMO into regenerating myotubes and rescue in laminin-α2 chain-null congenital muscular dystrophy mice.

Aoki Y, Nagata T, Yokota T, Nakamura A, Wood MJ, Partridge T, Takeda S. Aoki Y, et al. Hum Mol Genet. 2013 Dec 15;22(24):4914-28. doi: 10.1093/hmg/ddt341. Epub 2013 Jul 23. Hum Mol Genet. 2013. PMID: 23882132 Free PMC article.

7

Long-term efficacy of systemic multiexon skipping targeting dystrophin exons 45-55 with a cocktail of vivo-morpholinos in mdx52 mice.

Echigoya Y, Aoki Y, Miskew B, Panesar D, Touznik A, Nagata T, Tanihata J, Nakamura A, Nagaraju K, Yokota T. Echigoya Y, et al. Among authors: aoki y. Mol Ther Nucleic Acids. 2015 Feb 3;4(2):e225. doi: 10.1038/mtna.2014.76. Mol Ther Nucleic Acids. 2015. PMID: 25647512 Free PMC article.

8

Deletion of exons 3-9 encompassing a mutational hot spot in the DMD gene presents an asymptomatic phenotype, indicating a target region for multiexon skipping therapy.

Nakamura A, Fueki N, Shiba N, Motoki H, Miyazaki D, Nishizawa H, Echigoya Y, Yokota T, Aoki Y, Takeda S. Nakamura A, et al. Among authors: aoki y. J Hum Genet. 2016 Jul;61(7):663-7. doi: 10.1038/jhg.2016.28. Epub 2016 Mar 24. J Hum Genet. 2016. PMID: 27009627

9

Effects of systemic multiexon skipping with peptide-conjugated morpholinos in the heart of a dog model of Duchenne muscular dystrophy.

Echigoya Y, Nakamura A, Nagata T, Urasawa N, Lim KRQ, Trieu N, Panesar D, Kuraoka M, Moulton HM, Saito T, Aoki Y, Iversen P, Sazani P, Kole R, Maruyama R, Partridge T, Takeda S, Yokota T. Echigoya Y, et al. Among authors: aoki y. Proc Natl Acad Sci U S A. 2017 Apr 18;114(16):4213-4218. doi: 10.1073/pnas.1613203114. Epub 2017 Apr 3. Proc Natl Acad Sci U S A. 2017. PMID: 28373570 Free PMC article.

10

Exon Skipping Therapy Using Phosphorodiamidate Morpholino Oligomers in the mdx52 Mouse Model of Duchenne Muscular Dystrophy.

Miyatake S, Mizobe Y, Takizawa H, Hara Y, Yokota T, Takeda S, Aoki Y. Miyatake S, et al. Among authors: aoki y. Methods Mol Biol. 2018;1687:123-141. doi: 10.1007/978-1-4939-7374-3_9. Methods Mol Biol. 2018. PMID: 29067660

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