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Year | Number of Results |
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ERK5 and its role in tumour development.
Biochem Soc Trans. 2012 Feb;40(1):251-6. doi: 10.1042/BST20110663.
Biochem Soc Trans. 2012.
PMID: 22260700
Review.
Stimulating translational research: several European life science institutions put their heads together.
Bentires-Alj M, Rajan A, van Harten W, van Luenen HG, Kubicek S, Andersen JB, Saarela J, Cook SJ, Van Minnebruggen G, Roman-Roman S, Maurer C, Erler JT, Bertero MG.
Bentires-Alj M, et al.
Trends Mol Med. 2015 Sep;21(9):525-7. doi: 10.1016/j.molmed.2015.07.002. Epub 2015 Aug 5.
Trends Mol Med. 2015.
PMID: 26254816
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CDK1, not ERK1/2 or ERK5, is required for mitotic phosphorylation of BIMEL.
Gilley R, Lochhead PA, Balmanno K, Oxley D, Clark J, Cook SJ.
Gilley R, et al.
Cell Signal. 2012 Jan;24(1):170-80. doi: 10.1016/j.cellsig.2011.08.018. Epub 2011 Sep 8.
Cell Signal. 2012.
PMID: 21924351
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ERK1/2, but not ERK5, is necessary and sufficient for phosphorylation and activation of c-Fos.
Gilley R, March HN, Cook SJ.
Gilley R, et al.
Cell Signal. 2009 Jun;21(6):969-77. doi: 10.1016/j.cellsig.2009.02.006. Epub 2009 Feb 25.
Cell Signal. 2009.
PMID: 19249353
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Tumor cells with KRAS or BRAF mutations or ERK5/MAPK7 amplification are not addicted to ERK5 activity for cell proliferation.
Lochhead PA, Clark J, Wang LZ, Gilmour L, Squires M, Gilley R, Foxton C, Newell DR, Wedge SR, Cook SJ.
Lochhead PA, et al.
Cell Cycle. 2016;15(4):506-18. doi: 10.1080/15384101.2015.1120915.
Cell Cycle. 2016.
PMID: 26959608
Free PMC article.
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