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Year | Number of Results |
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Herpesvirus quiescence (QIF) in neuronal cells VI: Correlative analysis demonstrates usefulness of QIF-PC12 cells to examine HSV-1 latency, reactivation and genes implicated in its regulation.
Curr Eye Res. 2003 Mar-Apr;26(3-4):239-48. doi: 10.1076/ceyr.26.3.239.14901.
Curr Eye Res. 2003.
PMID: 12815553
Herpesvirus quiescence in neuronal cells IV: virus activation induced by pituitary adenylate cyclase-activating polypeptide (PACAP) involves the protein kinase A pathway.
Danaher RJ, Savells-Arb AD, Black SA Jr, Jacob RJ, Miller CS.
Danaher RJ, et al.
J Neurovirol. 2001 Apr;7(2):163-8. doi: 10.1080/13550280152058825.
J Neurovirol. 2001.
PMID: 11517389
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Herpesvirus quiescence in neuronal cells: antiviral conditions not required to establish and maintain HSV-2 quiescence.
Danaher RJ, Jacob RJ, Miller CS.
Danaher RJ, et al.
J Neurovirol. 2000 Aug;6(4):296-302. doi: 10.3109/13550280009030755.
J Neurovirol. 2000.
PMID: 10951553
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Heat stress activates production of herpes simplex virus type 1 from quiescently infected neurally differentiated PC12 cells.
Danaher RJ, Jacob RJ, Chorak MD, Freeman CS, Miller CS.
Danaher RJ, et al.
J Neurovirol. 1999 Aug;5(4):374-83. doi: 10.3109/13550289909029478.
J Neurovirol. 1999.
PMID: 10463859
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Establishment of a quiescent herpes simplex virus type 1 infection in neurally-differentiated PC12 cells.
Danaher RJ, Jacob RJ, Miller CS.
Danaher RJ, et al.
J Neurovirol. 1999 Jun;5(3):258-67. doi: 10.3109/13550289909015812.
J Neurovirol. 1999.
PMID: 10414516
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Molecular aspects of herpes simplex virus I latency, reactivation, and recurrence.
Miller CS, Danaher RJ, Jacob RJ.
Miller CS, et al.
Crit Rev Oral Biol Med. 1998;9(4):541-62. doi: 10.1177/10454411980090040901.
Crit Rev Oral Biol Med. 1998.
PMID: 9825226
Review.
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