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2006 1
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Germline gene fusions across species reveal the chromosomal instability regions and cancer susceptibility.
Zhou BW, Wu QQ, Mauki DH, Wang X, Zhang SR, Yin TT, Chen FL, Li C, Liu YH, Wang GD, Zhang YP. Zhou BW, et al. iScience. 2023 Nov 10;26(12):108431. doi: 10.1016/j.isci.2023.108431. eCollection 2023 Dec 15. iScience. 2023. PMID: 38205119 Free PMC article.
The canine transmissible venereal tumor (CTVT) is a clonal cell-mediated cancer with a long evolutionary history and extensive karyotype rearrangements in its genome. ...This study presents a comprehensive landscape of gene fusions (GFs) in CTVT, which offers a valuable ge …
The canine transmissible venereal tumor (CTVT) is a clonal cell-mediated cancer with a long evolutionary history and extensive karyot …
TPL-2 kinase induces phagosome acidification to promote macrophage killing of bacteria.
Breyer F, Härtlova A, Thurston T, Flynn HR, Chakravarty P, Janzen J, Peltier J, Heunis T, Snijders AP, Trost M, Ley SC. Breyer F, et al. EMBO J. 2021 May 17;40(10):e106188. doi: 10.15252/embj.2020106188. Epub 2021 Apr 21. EMBO J. 2021. PMID: 33881780 Free PMC article.
Quantitative proteomics revealed that blocking TPL-2 catalytic activity significantly altered the protein composition of phagosomes, particularly reducing the abundance of V-ATPase proton pump subunits. Furthermore, TPL-2 stimulated the phosphorylation of DMXL1, a regulato …
Quantitative proteomics revealed that blocking TPL-2 catalytic activity significantly altered the protein composition of phagosomes, particu …
Secondary leiomyosarcoma of the nasal cavity in a treated patient with possible hereditary retinoblastoma with germline reciprocal translocation of RB1 and DMXL1 and somatic TP53 mutation: A case report.
Yagi T, Nakamura H, Kukita Y, Wakamatsu T, Tamiya H, Nakai S, Watanabe M, Kakunaga S, Takami H, Suzuki R, Takenaka S, Hashii Y. Yagi T, et al. Mol Clin Oncol. 2023 Jul 10;19(2):65. doi: 10.3892/mco.2023.2661. eCollection 2023 Aug. Mol Clin Oncol. 2023. PMID: 37559881 Free PMC article.
The RNA panel sequencing detected a germline reciprocal translocation of RB1 and DMXL1, leading to a diagnosis of possible hereditary retinoblastoma. ...Further prospective investigations are necessary to fully understand the role of reciprocal translocation of RB1 and …
The RNA panel sequencing detected a germline reciprocal translocation of RB1 and DMXL1, leading to a diagnosis of possible hereditary …
Identification of novel genes associated with astrocytoma progression using suppression subtractive hybridization and real-time reverse transcription-polymerase chain reaction.
van den Boom J, Wolter M, Blaschke B, Knobbe CB, Reifenberger G. van den Boom J, et al. Int J Cancer. 2006 Nov 15;119(10):2330-8. doi: 10.1002/ijc.22108. Int J Cancer. 2006. PMID: 16865689 Free article.
These analyses revealed that 5 genes, i.e., AMOG (ATP1B2, 17p13.1), APOD (3q26.2-qter), DMXL1 (5q23.1) DRR1 (TU3A, 3p14.2) and PSD3 (KIAA09428/HCA67/EFA6R, 8p22), were expressed at significantly lower levels in secondary glioblastomas as compared to diffuse astrocytomas of …
These analyses revealed that 5 genes, i.e., AMOG (ATP1B2, 17p13.1), APOD (3q26.2-qter), DMXL1 (5q23.1) DRR1 (TU3A, 3p14.2) and PSD3 ( …
Gene profiling of growth factor independence 1B gene (Gfi-1B) in leukemic cells.
Koldehoff M, Zakrzewski JL, Klein-Hitpass L, Beelen DW, Elmaagacli AH. Koldehoff M, et al. Int J Hematol. 2008 Jan;87(1):39-47. doi: 10.1007/s12185-007-0013-z. Epub 2007 Dec 6. Int J Hematol. 2008. PMID: 18224412
Interestingly, transcripts within components of the signalling cascade of immune cells (PLD1, LAMP1, HSP90, IL6ST), of the tyrosine kinase pathway (TPR, RAC3) and of the transcription factors (RAC3, CEP290, JEM-1, ATR, MYC, SMC3, RARA, RBBP6) were found to be differentially expre …
Interestingly, transcripts within components of the signalling cascade of immune cells (PLD1, LAMP1, HSP90, IL6ST), of the tyrosine kinase p …
In Silico Improvement of beta3-peptide inhibitors of p53 x hDM2 and p53 x hDMX.
Michel J, Harker EA, Tirado-Rives J, Jorgensen WL, Schepartz A. Michel J, et al. J Am Chem Soc. 2009 May 13;131(18):6356-7. doi: 10.1021/ja901478e. J Am Chem Soc. 2009. PMID: 19415930 Free PMC article.
There is great interest in molecules capable of inhibiting the interactions between p53 and its negative regulators hDM2 and hDMX, as these molecules have validated potential against cancers in which one or both oncoproteins are overexpressed. We reported previously that a …
There is great interest in molecules capable of inhibiting the interactions between p53 and its negative regulators hDM2 and hDMX, as these …