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Ski drives an acute increase in MMP-9 gene expression and release in primary cardiac myofibroblasts.
Physiol Rep. 2018 Nov;6(22):e13897. doi: 10.14814/phy2.13897.
Physiol Rep. 2018.
PMID: 30488595
Free PMC article.
The Ski-Zeb2-Meox2 pathway provides a novel mechanism for regulation of the cardiac myofibroblast phenotype.
Cunnington RH, Northcott JM, Ghavami S, Filomeno KL, Jahan F, Kavosh MS, Davies JJ, Wigle JT, Dixon IM.
Cunnington RH, et al. Among authors: filomeno kl.
J Cell Sci. 2014 Jan 1;127(Pt 1):40-9. doi: 10.1242/jcs.126722. Epub 2013 Oct 23.
J Cell Sci. 2014.
PMID: 24155330
Item in Clipboard
SKI activates the Hippo pathway via LIMD1 to inhibit cardiac fibroblast activation.
Landry NM, Rattan SG, Filomeno KL, Meier TW, Meier SC, Foran SJ, Meier CF, Koleini N, Fandrich RR, Kardami E, Duhamel TA, Dixon IMC.
Landry NM, et al. Among authors: filomeno kl.
Basic Res Cardiol. 2021 Apr 13;116(1):25. doi: 10.1007/s00395-021-00865-9.
Basic Res Cardiol. 2021.
PMID: 33847835
Free PMC article.
Item in Clipboard
Autophagy is a regulator of TGF-β1-induced fibrogenesis in primary human atrial myofibroblasts.
Ghavami S, Cunnington RH, Gupta S, Yeganeh B, Filomeno KL, Freed DH, Chen S, Klonisch T, Halayko AJ, Ambrose E, Singal R, Dixon IM.
Ghavami S, et al. Among authors: filomeno kl.
Cell Death Dis. 2015 Mar 19;6(3):e1696. doi: 10.1038/cddis.2015.36.
Cell Death Dis. 2015.
PMID: 25789971
Free PMC article.
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Novel factors that activate and deactivate cardiac fibroblasts: A new perspective for treatment of cardiac fibrosis.
de Oliveira Camargo R, Abual'anaz B, Rattan SG, Filomeno KL, Dixon IMC.
de Oliveira Camargo R, et al. Among authors: filomeno kl.
Wound Repair Regen. 2021 Jul;29(4):667-677. doi: 10.1111/wrr.12947. Epub 2021 Jun 16.
Wound Repair Regen. 2021.
PMID: 34076932
Review.
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Intracellular signaling of cardiac fibroblasts.
Roche PL, Filomeno KL, Bagchi RA, Czubryt MP.
Roche PL, et al. Among authors: filomeno kl.
Compr Physiol. 2015 Apr;5(2):721-60. doi: 10.1002/cphy.c140044.
Compr Physiol. 2015.
PMID: 25880511
Review.
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