Ichinose A - Search Results

1

Inhibitors of Factor XIII/13 in older patients.

Ichinose A; Japanese Collaborative Research Group (JCRG) on AH13 Hemorrhagic Acquired Coagulopathies. Ichinose A, et al. Semin Thromb Hemost. 2014 Sep;40(6):704-11. doi: 10.1055/s-0034-1390151. Epub 2014 Sep 12. Semin Thromb Hemost. 2014. PMID: 25215637 Review.

2

Molecular approach to structure-function relationship of human coagulation factor XIII.

Ichinose A, Kaetsu H. Ichinose A, et al. Methods Enzymol. 1993;222:36-51. doi: 10.1016/0076-6879(93)22006-2. Methods Enzymol. 1993. PMID: 8412805 No abstract available.

3

Type I factor XIII deficiency is caused by a genetic defect of its b subunit: insertion of triplet AAC in exon III leads to premature termination in the second Sushi domain.

Izumi T, Hashiguchi T, Castaman G, Tosetto A, Rodeghiero F, Girolami A, Ichinose A. Izumi T, et al. Among authors: ichinose a. Blood. 1996 Apr 1;87(7):2769-74. Blood. 1996. PMID: 8639893 Free article.

4

The normal and abnormal genes of the a and b subunits in coagulation factor XIII.

Ichinose A, Izumi T, Hashiguchi T. Ichinose A, et al. Semin Thromb Hemost. 1996;22(5):385-91. doi: 10.1055/s-2007-999036. Semin Thromb Hemost. 1996. PMID: 8989821 Review.

5

Novel deletion and insertion mutations cause splicing defects, leading to severe reduction in mRNA levels of the A subunit in severe factor XIII deficiency.

Izumi T, Nagaoka U, Saito T, Takamatsu J, Saito H, Ichinose A. Izumi T, et al. Among authors: ichinose a. Thromb Haemost. 1998 Mar;79(3):479-85. Thromb Haemost. 1998. PMID: 9531026

6

Molecular mechanisms of type II factor XIII deficiency: novel Gly562-Arg mutation and C-terminal truncation of the A subunit cause factor XIII deficiency as characterized in a mammalian expression system.

Takahashi N, Tsukamoto H, Umeyama H, Castaman G, Rodeghiero F, Ichinose A. Takahashi N, et al. Among authors: ichinose a. Blood. 1998 Apr 15;91(8):2830-8. Blood. 1998. PMID: 9531593 Free article.

7

Arg260-Cys mutation in severe factor XIII deficiency: conformational change of the A subunit is predicted by molecular modelling and mechanics.

Ichinose A, Tsukamoto H, Izumi T, Yamazaki T, Togashi M, Takamatsu J, Saito H, Umeyama H. Ichinose A, et al. Br J Haematol. 1998 May;101(2):264-72. doi: 10.1046/j.1365-2141.1998.00698.x. Br J Haematol. 1998. PMID: 9609521 Free article.

8

A founder effect is proposed for factor XIII B subunit deficiency caused by the insertion of triplet AAC in exon III encoding the second Sushi domain.

Souri M, Izumi T, Higashi Y, Girolami A, Ichinose A. Souri M, et al. Among authors: ichinose a. Thromb Haemost. 1998 Aug;80(2):211-3. Thromb Haemost. 1998. PMID: 9716138

9

[Deficiency of A or B subunit for coagulation factor XIII].

Souri M, Ichinose A. Souri M, et al. Among authors: ichinose a. Ryoikibetsu Shokogun Shirizu. 1998;(21 Pt 2):464-7. Ryoikibetsu Shokogun Shirizu. 1998. PMID: 9833543 Review. Japanese. No abstract available.

10

Transcriptional regulation of cell type-specific expression of the TATA-less A subunit gene for human coagulation factor XIII.

Kida M, Souri M, Yamamoto M, Saito H, Ichinose A. Kida M, et al. Among authors: ichinose a. J Biol Chem. 1999 Mar 5;274(10):6138-47. doi: 10.1074/jbc.274.10.6138. J Biol Chem. 1999. PMID: 10037697 Free article.

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