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Normal and defective expression of the thyroglobulin gene.
Vassart G, Brocas H, Christophe D, de Martynoff G, Gérard C, Hansen C, Juvenal G, Ludgate M, Mercken L, Parma J, et al. Vassart G, et al. Among authors: parma j. Horm Res. 1987;26(1-4):8-11. doi: 10.1159/000180680. Horm Res. 1987. PMID: 3297966 Review.
Structure, expression and regulation of the thyroglobulin gene.
Vassart G, Bacolla A, Brocas H, Christophe D, de Martynoff G, Leriche A, Mercken L, Parma J, Pohl V, Targovnik H, et al. Vassart G, et al. Among authors: parma j. Mol Cell Endocrinol. 1985 May;40(2-3):89-97. doi: 10.1016/0303-7207(85)90162-5. Mol Cell Endocrinol. 1985. PMID: 3891458 Review. No abstract available.
Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.
Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Mol Endocrinol. 1995 Jun;9(6):725-33. doi: 10.1210/mend.9.6.8592518. Mol Endocrinol. 1995. PMID: 8592518
Somatic and germline mutations of the TSH receptor gene in thyroid diseases.
Van Sande J, Parma J, Tonacchera M, Swillens S, Dumont J, Vassart G. Van Sande J, et al. Among authors: parma j. J Clin Endocrinol Metab. 1995 Sep;80(9):2577-85. doi: 10.1210/jcem.80.9.7673398. J Clin Endocrinol Metab. 1995. PMID: 7673398 Review. No abstract available.
Constitutively active receptors as a disease-causing mechanism.
Parma J, Duprez L, Van Sande J, Paschke R, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Mol Cell Endocrinol. 1994 Apr;100(1-2):159-62. doi: 10.1016/0303-7207(94)90296-8. Mol Cell Endocrinol. 1994. PMID: 8056151 Review.
Molecular genetics of thyroid diseases.
Ledent C, Parma J, Dumont J, Vassart G, Targovnik H. Ledent C, et al. Among authors: parma j. Eur J Endocrinol. 1994 Jan;130(1):8-14. doi: 10.1530/eje.0.1300008. Eur J Endocrinol. 1994. PMID: 8124482 Review.
89 results