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Page 1
Functional characterization of a novel KCNJ11 in frame mutation-deletion associated with infancy-onset diabetes and a mild form of intermediate DEND: a battle between K(ATP) gain of channel activity and loss of channel expression.
Lin YW, Li A, Grasso V, Battaglia D, Crinò A, Colombo C, Barbetti F, Nichols CG. Lin YW, et al. PLoS One. 2013 May 7;8(5):e63758. doi: 10.1371/journal.pone.0063758. Print 2013. PLoS One. 2013. PMID: 23667671 Free PMC article.
The diabetic β-cell: hyperstimulated vs. hyperexcited.
Nichols CG, Remedi MS. Nichols CG, et al. Diabetes Obes Metab. 2012 Oct;14 Suppl 3(0 3):129-35. doi: 10.1111/j.1463-1326.2012.01655.x. Diabetes Obes Metab. 2012. PMID: 22928573 Free PMC article. Review.
Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 (KCNJ11) mutation V290M mutation: K(ATP) channel inactivation mechanism and clinical management.
Loechner KJ, Akrouh A, Kurata HT, Dionisi-Vici C, Maiorana A, Pizzoferro M, Rufini V, de Ville de Goyet J, Colombo C, Barbetti F, Koster JC, Nichols CG. Loechner KJ, et al. Diabetes. 2011 Jan;60(1):209-17. doi: 10.2337/db10-0731. Epub 2010 Oct 27. Diabetes. 2011. PMID: 20980454 Free PMC article.
19 results