Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2013 1
2017 2
2021 3
2022 1
2023 1
2024 0

Text availability

Article attribute

Article type

Publication date

Search Results

8 results

Results by year

Filters applied: . Clear all
Page 1
SNTB1 regulates colorectal cancer cell proliferation and metastasis through YAP1 and the WNT/beta-catenin pathway.
Zhang H, Li Z, Jiang J, Lei Y, Xie J, Liu Y, Yi B. Zhang H, et al. Cell Cycle. 2023 Sep;22(17):1865-1883. doi: 10.1080/15384101.2023.2244778. Epub 2023 Aug 17. Cell Cycle. 2023. PMID: 37592763
SNTB1 positively regulated Yes1 associated transcriptional regulator (YAP1) expression; YAP1 partially reversed the effects of SNTB1 on colorectal cancer cell phenotypes and the Wnt/beta-catenin/MYC signaling. In conclusion, SNTB1 knockdown inhibits co
SNTB1 positively regulated Yes1 associated transcriptional regulator (YAP1) expression; YAP1 partially reversed the effects of SNT
Beta-1 syntrophin (SNTB1) regulates colorectal cancer progression and stemness via regulation of the Wnt/beta-catenin signaling pathway.
Liang Y, Wang B, Chen S, Ye Z, Chai X, Li R, Li X, Kong G, Li Y, Zhang X, Che Z, Xie Q, Lian J, Lin B, Zhang X, Huang X, Huang W, Qiu X, Zeng J. Liang Y, et al. Ann Transl Med. 2021 Jun;9(12):1016. doi: 10.21037/atm-21-2700. Ann Transl Med. 2021. PMID: 34277816 Free PMC article.
However, the physiological role of SNTB1 is poorly understood. This study aims to explore the role of SNTB1 in colorectal cancer (CRC) tumorigenesis and progression, with particular focus on SNTB1's expression pattern, clinical relevance, and possible …
However, the physiological role of SNTB1 is poorly understood. This study aims to explore the role of SNTB1 in colorectal c
Upregulation of SNTB1 correlates with poor prognosis and promotes cell growth by negative regulating PKN2 in colorectal cancer.
Liu L, Chen Y, Lin X, Wu M, Li J, Xie Q, Sferra TJ, Han Y, Liu H, Cao L, Yao M, Peng J, Shen A. Liu L, et al. Cancer Cell Int. 2021 Oct 18;21(1):547. doi: 10.1186/s12935-021-02246-7. Cancer Cell Int. 2021. PMID: 34663329 Free PMC article.
A preliminary study identified syntrophin beta 1 (SNTB1) as a potential oncogene in CRC. However, the clinical significance, biological function, and underlying mechanisms of SNTB1 in CRC remain largely unknown. ...CONCLUSIONS: These findings indicate that SNTB1
A preliminary study identified syntrophin beta 1 (SNTB1) as a potential oncogene in CRC. However, the clinical significance, biologic …
Single nucleotide polymorphisms in an Indian cohort and association of CNTN4, MMP2 and SNTB1 variants with oral cancer.
Yete S, Pradhan S, Saranath D. Yete S, et al. Cancer Genet. 2017 Aug;214-215:16-25. doi: 10.1016/j.cancergen.2017.03.006. Epub 2017 Mar 23. Cancer Genet. 2017. PMID: 28595731
Oral cancer is a high incidence cancer in India primarily due to the prevalent tobacco/areca nut chewing habits and hence a major health concern. India constitutes 26% of the global oral cancer burden. Besides the well-established risk factors, the genomic co …
Oral cancer is a high incidence cancer in India primarily due to the prevalent tobacco/areca nut chewing habits and hence a ma …
OCT1-target neural gene PFN2 promotes tumor growth in androgen receptor-negative prostate cancer.
Obinata D, Funakoshi D, Takayama K, Hara M, Niranjan B, Teng L, Lawrence MG, Taylor RA, Risbridger GP, Suzuki Y, Takahashi S, Inoue S. Obinata D, et al. Sci Rep. 2022 Apr 12;12(1):6094. doi: 10.1038/s41598-022-10099-x. Sci Rep. 2022. PMID: 35413990 Free PMC article.
Androgen and androgen receptor (AR) targeted therapies are the main treatment for most prostate cancer (PC) patients. Although AR signaling inhibitors are effective, tumors can evade this treatment by transforming to an AR-negative PC via lineage plasticity. ...Immunohisto …
Androgen and androgen receptor (AR) targeted therapies are the main treatment for most prostate cancer (PC) patients. Although AR sig …
Identification and Characterization of Non-Coding RNAs in Thymoma.
Ji G, Ren R, Fang X. Ji G, et al. Med Sci Monit. 2021 Jul 5;27:e929727. doi: 10.12659/MSM.929727. Med Sci Monit. 2021. PMID: 34219124 Free PMC article.
RESULTS We found 21 fusion genes, including KMT2A-MAML2, HADHB-REEP1, COQ3-CGA, MCM4-SNTB1, and IFT140-ACTN4, as the most frequent and significant in thymomas. ...
RESULTS We found 21 fusion genes, including KMT2A-MAML2, HADHB-REEP1, COQ3-CGA, MCM4-SNTB1, and IFT140-ACTN4, as the most frequent an …
Characterization of genetic aberrations in a single case of metastatic thymic adenocarcinoma.
Lee Y, Park S, Lee SH, Lee H. Lee Y, et al. BMC Cancer. 2017 May 15;17(1):330. doi: 10.1186/s12885-017-3282-9. BMC Cancer. 2017. PMID: 28506304 Free PMC article.
Copy number analysis revealed a complex chromosomal rearrangement of chromosome 8, which resulted in gene fusion between MCM4 and SNTB1 and dramatic amplification of MYC and NDRG1. Focal deletion was detected at human leukocyte antigen (HLA) class II alleles, which was pre …
Copy number analysis revealed a complex chromosomal rearrangement of chromosome 8, which resulted in gene fusion between MCM4 and SNTB1
Gene expression signature of non-involved lung tissue associated with survival in lung adenocarcinoma patients.
Galvan A, Frullanti E, Anderlini M, Manenti G, Noci S, Dugo M, Ambrogi F, De Cecco L, Spinelli R, Piazza R, Pirola A, Gambacorti-Passerini C, Incarbone M, Alloisio M, Tosi D, Nosotti M, Santambrogio L, Pastorino U, Dragani TA. Galvan A, et al. Carcinogenesis. 2013 Dec;34(12):2767-73. doi: 10.1093/carcin/bgt294. Epub 2013 Aug 25. Carcinogenesis. 2013. PMID: 23978379
These clinical discrepancies may be due to the genetic background modulating an individual's predisposition to fighting cancer. Herein, we hypothesized that the lung microenvironment, as reflected by its expression profile, may affect lung adenocarcinoma patients' survival …
These clinical discrepancies may be due to the genetic background modulating an individual's predisposition to fighting cancer. Herei …