Shah SD - Search Results

1

Role of epinephrine-mediated beta-adrenergic mechanisms in hypoglycemic glucose counterregulation and posthypoglycemic hyperglycemia in insulin-dependent diabetes mellitus.

Popp DA, Shah SD, Cryer PE. Popp DA, et al. Among authors: shah sd. J Clin Invest. 1982 Feb;69(2):315-26. doi: 10.1172/jci110455. J Clin Invest. 1982. PMID: 6120181 Free PMC article.

2

Increased fat and skeletal muscle beta-adrenergic receptors but unaltered metabolic and hemodynamic sensitivity to epinephrine in vivo in experimental human thyrotoxicosis.

Liggett SB, Shah SD, Cryer PE. Liggett SB, et al. Among authors: shah sd. J Clin Invest. 1989 Mar;83(3):803-9. doi: 10.1172/JCI113961. J Clin Invest. 1989. PMID: 2537849 Free PMC article.

3

Direct relationship between mononuclear leukocyte and lung beta-adrenergic receptors and apparent reciprocal regulation of extravascular, but not intravascular, alpha- and beta-adrenergic receptors by the sympathochromaffin system in humans.

Liggett SB, Marker JC, Shah SD, Roper CL, Cryer PE. Liggett SB, et al. Among authors: shah sd. J Clin Invest. 1988 Jul;82(1):48-56. doi: 10.1172/JCI113600. J Clin Invest. 1988. PMID: 2839552 Free PMC article.

4

Glucoregulation during exercise: hypoglycemia is prevented by redundant glucoregulatory systems, sympathochromaffin activation, and changes in islet hormone secretion.

Hoelzer DR, Dalsky GP, Clutter WE, Shah SD, Holloszy JO, Cryer PE. Hoelzer DR, et al. Among authors: shah sd. J Clin Invest. 1986 Jan;77(1):212-21. doi: 10.1172/JCI112279. J Clin Invest. 1986. PMID: 3511090 Free PMC article. Retracted.

5

Glycemic thresholds for activation of glucose counterregulatory systems are higher than the threshold for symptoms.

Schwartz NS, Clutter WE, Shah SD, Cryer PE. Schwartz NS, et al. Among authors: shah sd. J Clin Invest. 1987 Mar;79(3):777-81. doi: 10.1172/JCI112884. J Clin Invest. 1987. PMID: 3546378 Free PMC article.

6

Enhanced glycemic responsiveness to epinephrine in insulin-dependent diabetes mellitus is the result of the inability to secrete insulin. Augmented insulin secretion normally limits the glycemic, but not the lipolytic or ketogenic, response to epinephrine in humans.

Berk MA, Clutter WE, Skor D, Shah SD, Gingerich RP, Parvin CA, Cryer PE. Berk MA, et al. Among authors: shah sd. J Clin Invest. 1985 Jun;75(6):1842-51. doi: 10.1172/JCI111898. J Clin Invest. 1985. PMID: 3891786 Free PMC article.

7

Mechanisms of postprandial glucose counterregulation in man. Physiologic roles of glucagon and epinephrine vis-a-vis insulin in the prevention of hypoglycemia late after glucose ingestion.

Tse TF, Clutter WE, Shah SD, Cryer PE. Tse TF, et al. Among authors: shah sd. J Clin Invest. 1983 Jul;72(1):278-86. doi: 10.1172/jci110967. J Clin Invest. 1983. PMID: 6135707 Free PMC article.

8

Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man.

Rosen SG, Clutter WE, Berk MA, Shah SD, Cryer PE. Rosen SG, et al. Among authors: shah sd. J Clin Invest. 1984 Feb;73(2):405-11. doi: 10.1172/JCI111226. J Clin Invest. 1984. PMID: 6142057 Free PMC article.

9

Triiodothyronine-induced thyrotoxicosis increases mononuclear leukocyte beta-adrenergic receptor density in man.

Ginsberg AM, Clutter WE, Shah SD, Cryer PE. Ginsberg AM, et al. Among authors: shah sd. J Clin Invest. 1981 Jun;67(6):1785-91. doi: 10.1172/jci110218. J Clin Invest. 1981. PMID: 6263953 Free PMC article.

10

Neuroendocrine responses to glucose ingestion in man. Specificity, temporal relationships, and quantitative aspects.

Tse TF, Clutter WE, Shah SD, Miller JP, Cryer PE. Tse TF, et al. Among authors: shah sd. J Clin Invest. 1983 Jul;72(1):270-7. doi: 10.1172/jci110966. J Clin Invest. 1983. PMID: 6409929 Free PMC article.

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