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1998 1
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90 results

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Page 1
SMARCD1 is a transcriptional target of specific non-hotspot mutant p53 forms.
Adduri RSR, George SA, Kavadipula P, Bashyam MD. Adduri RSR, et al. J Cell Physiol. 2020 May;235(5):4559-4570. doi: 10.1002/jcp.29332. Epub 2019 Oct 21. J Cell Physiol. 2020. PMID: 31637714
Increased SMARCD1 expression predicted poor survival in HNSCC tumors harboring missense p53 mutations. Our results suggest SMARCD1 to be a novel transcriptional target of mutant p53....
Increased SMARCD1 expression predicted poor survival in HNSCC tumors harboring missense p53 mutations. Our results suggest SMARCD1 …
Smarcd1 Inhibits the Malignant Phenotypes of Human Glioblastoma Cells via Crosstalk with Notch1.
Zhu Y, Wang H, Fei M, Tang T, Niu W, Zhang L. Zhu Y, et al. Mol Neurobiol. 2021 Apr;58(4):1438-1452. doi: 10.1007/s12035-020-02190-z. Epub 2020 Nov 14. Mol Neurobiol. 2021. PMID: 33190170 Free PMC article.
Importantly, the depletion of Smarcd1 promoted cell proliferation, invasion, and chemoresistance, whereas enhanced expression of Smarcd1 inhibited tumor-malignant phenotypes. Mechanistic research demonstrated that overexpression of Smarcd1 decreased the expre …
Importantly, the depletion of Smarcd1 promoted cell proliferation, invasion, and chemoresistance, whereas enhanced expression of S …
Smarcd1 antagonizes the apoptosis of injured MES23.5 DA cells by enhancing the effect of Six2 on GDNF expression.
Gao J, Qin DL, Tang CX, Kang XY, Song CJ, Zhang CT. Gao J, et al. Neurosci Lett. 2021 Aug 24;760:136088. doi: 10.1016/j.neulet.2021.136088. Epub 2021 Jul 4. Neurosci Lett. 2021. PMID: 34233203
Our results confirmed that Smarcd1 formed a transcription complex with Six2, and Smarcd1 mainly binded to the 2840 bp-2933 bp region of the GDNF promoter. Furthermore, knockdown of Smarcd1 inhibited the effect of Six2 on GDNF expression, and resulted in decre …
Our results confirmed that Smarcd1 formed a transcription complex with Six2, and Smarcd1 mainly binded to the 2840 bp-2933 bp …
SMARCD1 regulates senescence-associated lipid accumulation in hepatocytes.
Inoue C, Zhao C, Tsuduki Y, Udono M, Wang L, Nomura M, Katakura Y. Inoue C, et al. NPJ Aging Mech Dis. 2017 Aug 30;3:11. doi: 10.1038/s41514-017-0011-1. eCollection 2017. NPJ Aging Mech Dis. 2017. PMID: 28868154 Free PMC article.
We demonstrated that the reduced expression of SMARCD1 triggers cellular senescence and induces the accumulation of lipids, suggesting that SMARCD1 acts as a mediator in these processes. ...The results obtained here suggest that dietary nutrient-associated impaired …
We demonstrated that the reduced expression of SMARCD1 triggers cellular senescence and induces the accumulation of lipids, suggestin …
SMARCD1 is a "Goldilocks" metastasis modifier.
Ross C, Gong LY, Jenkins LM, Ha NH, Majocha M, Hunter K. Ross C, et al. bioRxiv [Preprint]. 2024 Jan 29:2024.01.24.577061. doi: 10.1101/2024.01.24.577061. bioRxiv. 2024. PMID: 38410477 Free PMC article. Preprint.
Using cell lines, 3D culture, mouse models, and clinical data, we pinpoint Smarcd1 mRNA as a key target of all three RNA-BPs. Strikingly, both high and low expression of Smarcd1 is associated with positive clinical outcomes, while intermediate expression significant …
Using cell lines, 3D culture, mouse models, and clinical data, we pinpoint Smarcd1 mRNA as a key target of all three RNA-BPs. Strikin …
Integrated analysis reveals SMARCD1 is a potential biomarker and therapeutic target in skin cutaneous melanoma.
Chen J, Yu N, Ou S, Wang X, Li H, Zhu H. Chen J, et al. J Cancer Res Clin Oncol. 2023 Oct;149(13):11619-11634. doi: 10.1007/s00432-023-05064-8. Epub 2023 Jul 4. J Cancer Res Clin Oncol. 2023. PMID: 37401939
Then we utilized immunohistochemical staining to measure the SMARCD1 expression in both SKCM tissues and normal skin tissues. Furthermore, we conducted in vitro experimentation to evaluate the effects of SMARCD1 knockdown on SKCM cells. ...SMARCD1 is likely i …
Then we utilized immunohistochemical staining to measure the SMARCD1 expression in both SKCM tissues and normal skin tissues. Further …
Enhanced SMARCD1, a subunit of the SWI/SNF complex, promotes liver cancer growth through the mTOR pathway.
Zhou Y, Xu Q, Tao L, Chen Y, Shu Y, Wu Z, Lu C, Shi Y, Bu H. Zhou Y, et al. Clin Sci (Lond). 2020 Jun 26;134(12):1457-1472. doi: 10.1042/CS20200244. Clin Sci (Lond). 2020. PMID: 32514535
We further conducted in vitro and in vivo experiments and revealed that SMARCD1 promotes liver cancer growth by activating the mTOR signaling pathway. In conclusion, our study has revealed that the expression of SWI/SNF complex subunits, especially SMARCD1, is highl …
We further conducted in vitro and in vivo experiments and revealed that SMARCD1 promotes liver cancer growth by activating the mTOR s …
microRNA-99a-5p induces cellular senescence in gemcitabine-resistant bladder cancer by targeting SMARCD1.
Tamai M, Tatarano S, Okamura S, Fukumoto W, Kawakami I, Osako Y, Sakaguchi T, Sugita S, Yonemori M, Yamada Y, Nakagawa M, Enokida H, Yoshino H. Tamai M, et al. Mol Oncol. 2022 Mar;16(6):1329-1346. doi: 10.1002/1878-0261.13192. Epub 2022 Feb 28. Mol Oncol. 2022. PMID: 35148461 Free PMC article.
RNA sequencing analysis was performed to find the target gene of miR-99a-5p. SMARCD1 was selected as a candidate gene. Dual-luciferase reporter assays showed that miR-99a-5p directly regulated SMARCD1. ...Furthermore, beta-galactosidase staining showed that miR-99a- …
RNA sequencing analysis was performed to find the target gene of miR-99a-5p. SMARCD1 was selected as a candidate gene. Dual-luciferas …
Expression of SMARCD1 interacts with age in association with asthma control on inhaled corticosteroid therapy.
McGeachie MJ, Sordillo JE, Dahlin A, Wang AL, Lutz SM, Tantisira KG, Panganiban R, Lu Q, Sajuthi S, Urbanek C, Kelly R, Saef B, Eng C, Oh SS, Kho AT, Croteau-Chonka DC, Weiss ST, Raby BA, Mak ACY, Rodriguez-Santana JR, Burchard EG, Seibold MA, Wu AC. McGeachie MJ, et al. Respir Res. 2020 Jan 28;21(1):31. doi: 10.1186/s12931-020-1295-4. Respir Res. 2020. PMID: 31992292 Free PMC article.
This result replicated in GALA II (p = 3.8 10(- 8)). Cellular assays confirmed the role of SMARCD1 in glucocorticoid response in airway epithelial cells. CONCLUSION: Focusing on age-dependent factors may help identify novel indicators of asthma medication response. Age app …
This result replicated in GALA II (p = 3.8 10(- 8)). Cellular assays confirmed the role of SMARCD1 in glucocorticoid response in airw …
90 results