Williams TA - Search Results

1

Whole blood methylome-derived features to discriminate endocrine hypertension.

Armignacco R, Reel PS, Reel S, Jouinot A, Septier A, Gaspar C, Perlemoine K, Larsen CK, Bouys L, Braun L, Riester A, Kroiss M, Bonnet-Serrano F, Amar L, Blanchard A, Gimenez-Roqueplo AP, Prejbisz A, Januszewicz A, Dobrowolski P, Davies E, MacKenzie SM, Rossi GP, Lenzini L, Ceccato F, Scaroni C, Mulatero P, Williams TA, Pecori A, Monticone S, Beuschlein F, Reincke M, Zennaro MC, Bertherat J, Jefferson E, Assié G. Armignacco R, et al. Among authors: williams ta. Clin Epigenetics. 2022 Nov 3;14(1):142. doi: 10.1186/s13148-022-01347-y. Clin Epigenetics. 2022. PMID: 36329530 Free PMC article.

2

Blood pressure in patients with primary aldosteronism is influenced by bradykinin B(2) receptor and alpha-adducin gene polymorphisms.

Mulatero P, Williams TA, Milan A, Paglieri C, Rabbia F, Fallo F, Veglio F. Mulatero P, et al. Among authors: williams ta. J Clin Endocrinol Metab. 2002 Jul;87(7):3337-43. doi: 10.1210/jcem.87.7.8666. J Clin Endocrinol Metab. 2002. PMID: 12107246

3

Bradykinin B2 receptor gene (-58T/C) polymorphism influences baroreflex sensitivity in never-treated hypertensive patients.

Milan A, Mulatero P, Williams TA, Carra R, Schiavone D, Martuzzi R, Rabbia F, Veglio F. Milan A, et al. Among authors: williams ta. J Hypertens. 2005 Jan;23(1):63-9. doi: 10.1097/00004872-200501000-00014. J Hypertens. 2005. PMID: 15643126

4

Role of HSD11B2 polymorphisms in essential hypertension and the diuretic response to thiazides.

Williams TA, Mulatero P, Filigheddu F, Troffa C, Milan A, Argiolas G, Parpaglia PP, Veglio F, Glorioso N. Williams TA, et al. Kidney Int. 2005 Feb;67(2):631-7. doi: 10.1111/j.1523-1755.2005.67119.x. Kidney Int. 2005. PMID: 15673310 Free article.

5

Protective effect of spironolactone on endothelial cell apoptosis.

Williams TA, Verhovez A, Milan A, Veglio F, Mulatero P. Williams TA, et al. Endocrinology. 2006 May;147(5):2496-505. doi: 10.1210/en.2005-1318. Epub 2006 Feb 23. Endocrinology. 2006. PMID: 16497808

6

Teratocarcinoma-derived growth factor-1 is upregulated in aldosterone-producing adenomas and increases aldosterone secretion and inhibits apoptosis in vitro.

Williams TA, Monticone S, Morello F, Liew CC, Mengozzi G, Pilon C, Asioli S, Sapino A, Veglio F, Mulatero P. Williams TA, et al. Hypertension. 2010 Jun;55(6):1468-75. doi: 10.1161/HYPERTENSIONAHA.110.150318. Epub 2010 Apr 12. Hypertension. 2010. PMID: 20385969 Free article.

7

a Novel Y152C KCNJ5 mutation responsible for familial hyperaldosteronism type III.

Monticone S, Hattangady NG, Penton D, Isales CM, Edwards MA, Williams TA, Sterner C, Warth R, Mulatero P, Rainey WE. Monticone S, et al. Among authors: williams ta. J Clin Endocrinol Metab. 2013 Nov;98(11):E1861-5. doi: 10.1210/jc.2013-2428. Epub 2013 Sep 13. J Clin Endocrinol Metab. 2013. PMID: 24037882 Free PMC article.

8

Understanding primary aldosteronism: impact of next generation sequencing and expression profiling.

Monticone S, Else T, Mulatero P, Williams TA, Rainey WE. Monticone S, et al. Among authors: williams ta. Mol Cell Endocrinol. 2015 Jan 5;399:311-20. doi: 10.1016/j.mce.2014.09.015. Epub 2014 Sep 18. Mol Cell Endocrinol. 2015. PMID: 25240470 Free PMC article. Review.

9

A case of severe hyperaldosteronism caused by a de novo mutation affecting a critical salt bridge Kir3.4 residue.

Monticone S, Bandulik S, Stindl J, Zilbermint M, Dedov I, Mulatero P, Allgaeuer M, Lee CC, Stratakis CA, Williams TA, Tiulpakov A. Monticone S, et al. Among authors: williams ta. J Clin Endocrinol Metab. 2015 Jan;100(1):E114-8. doi: 10.1210/jc.2014-3636. J Clin Endocrinol Metab. 2015. PMID: 25322277 Free PMC article.

10

KCNJ5 mutations are the most frequent genetic alteration in primary aldosteronism.

Williams TA, Monticone S, Mulatero P. Williams TA, et al. Hypertension. 2015 Mar;65(3):507-9. doi: 10.1161/HYPERTENSIONAHA.114.04636. Epub 2015 Jan 26. Hypertension. 2015. PMID: 25624337 Free article. No abstract available.

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