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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1956 1
1958 1
1959 1
1960 1
1961 1
1962 1
1963 1
1964 1
1965 3
1966 2
1967 4
1968 2
1969 2
1970 5
1971 2
1972 1
1973 2
1974 1
1975 6
1976 2
1977 1
1978 3
1979 8
1980 5
1981 10
1982 1
1983 4
1984 7
1985 6
1986 6
1987 6
1988 9
1989 11
1990 15
1991 15
1992 15
1993 10
1994 10
1995 25
1996 15
1997 8
1998 16
1999 16
2000 10
2001 9
2002 10
2003 13
2004 12
2005 18
2006 14
2007 12
2008 14
2009 12
2010 13
2011 14
2012 15
2013 15
2014 11
2015 5
2016 23
2017 13
2018 16
2019 12
2020 17
2021 24
2022 18
2023 22
2024 5

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581 results

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Page 1
Lactobacillus rhamnosus AD3 as a Promising Alternative for Probiotic Products.
Stivala A, Carota G, Fuochi V, Furneri PM. Stivala A, et al. Biomolecules. 2021 Jan 13;11(1):94. doi: 10.3390/biom11010094. Biomolecules. 2021. PMID: 33450875 Free PMC article.
The strains belonged to the species of L. crispatus, L. gasseri, L. rhamnosus and L. delbruckii. Among these, a strain of L. rhamnosus named AD3 showed the best probiotic properties. As probiotics are already in use in many clinical practice and there are no major safety c …
The strains belonged to the species of L. crispatus, L. gasseri, L. rhamnosus and L. delbruckii. Among these, a strain of L. rhamnosus named …
Ad3-hTERT-E1A, a fully serotype 3 oncolytic adenovirus, in patients with chemotherapy refractory cancer.
Hemminki O, Diaconu I, Cerullo V, Pesonen SK, Kanerva A, Joensuu T, Kairemo K, Laasonen L, Partanen K, Kangasniemi L, Lieber A, Pesonen S, Hemminki A. Hemminki O, et al. Mol Ther. 2012 Sep;20(9):1821-30. doi: 10.1038/mt.2012.115. Epub 2012 Aug 7. Mol Ther. 2012. PMID: 22871667 Free PMC article.
Twenty-five patients with chemotherapy refractory cancer were treated with a fully serotype 3-based oncolytic adenovirus Ad3-hTERT-E1A. In mice, Ad3 induced higher amounts of cytokines but less liver damage than Ad5 or Ad5/3. ...Patients that had been previously tre …
Twenty-five patients with chemotherapy refractory cancer were treated with a fully serotype 3-based oncolytic adenovirus Ad3-hTERT-E1 …
Efficacy of downtitration or treatment withdrawal compared with continuous dosing after successful treatment with baricitinib in patients with moderate-to-severe atopic dermatitis in a randomized substudy from the long-term extension study BREEZE-AD3.
Reich K, Simpson E, Wollenberg A, Bissonnette R, Abe M, Cardillo T, Janes J, Sun L, Chen S, Silverberg JI. Reich K, et al. Br J Dermatol. 2023 Feb 10;188(2):208-217. doi: 10.1093/bjd/ljac057. Br J Dermatol. 2023. PMID: 36763879 Clinical Trial.
BACKGROUND: Baricitinib, an oral, selective Janus kinase 1/2 inhibitor, demonstrated long-term efficacy in moderate-to-severe atopic dermatitis in an ongoing double-blind, phase III, long-term extension study, BREEZE-AD3 (NCT03334435). OBJECTIVES: To evaluate the efficacy …
BACKGROUND: Baricitinib, an oral, selective Janus kinase 1/2 inhibitor, demonstrated long-term efficacy in moderate-to-severe atopic dermati …
Long-term Efficacy of Baricitinib in Adults With Moderate to Severe Atopic Dermatitis Who Were Treatment Responders or Partial Responders: An Extension Study of 2 Randomized Clinical Trials.
Silverberg JI, Simpson EL, Wollenberg A, Bissonnette R, Kabashima K, DeLozier AM, Sun L, Cardillo T, Nunes FP, Reich K. Silverberg JI, et al. JAMA Dermatol. 2021 Jun 1;157(6):691-699. doi: 10.1001/jamadermatol.2021.1273. JAMA Dermatol. 2021. PMID: 33978711 Free PMC article. Clinical Trial.
DESIGN, SETTING, AND PARTICIPANTS: Patients completing BREEZE-AD1/BREEZE-AD2 entered the ongoing, multicenter, double-blind, long-term extension study BREEZE-AD3. The study was initiated on March 28, 2018. Data were analyzed on December 13, 2019. ...Itch data were collecte …
DESIGN, SETTING, AND PARTICIPANTS: Patients completing BREEZE-AD1/BREEZE-AD2 entered the ongoing, multicenter, double-blind, long-term exten …
The structural basis for the integrity of adenovirus Ad3 dodecahedron.
Szolajska E, Burmeister WP, Zochowska M, Nerlo B, Andreev I, Schoehn G, Andrieu JP, Fender P, Naskalska A, Zubieta C, Cusack S, Chroboczek J. Szolajska E, et al. PLoS One. 2012;7(9):e46075. doi: 10.1371/journal.pone.0046075. Epub 2012 Sep 25. PLoS One. 2012. PMID: 23049939 Free PMC article.
During the viral life cycle adenoviruses produce excess capsid proteins. Human adenovirus serotype 3 (Ad3) synthesizes predominantly an excess of free pentons, the complexes of pentameric penton base and trimeric fiber proteins, which are responsible for virus penetration. …
During the viral life cycle adenoviruses produce excess capsid proteins. Human adenovirus serotype 3 (Ad3) synthesizes predominantly …
Intracellular Signaling and Desmoglein 2 Shedding Triggered by Human Adenoviruses Ad3, Ad14, and Ad14P1.
Wang H, Ducournau C, Saydaminova K, Richter M, Yumul R, Ho M, Carter D, Zubieta C, Fender P, Lieber A. Wang H, et al. J Virol. 2015 Nov;89(21):10841-59. doi: 10.1128/JVI.01425-15. Epub 2015 Aug 19. J Virol. 2015. PMID: 26292319 Free PMC article.
We recently discovered that desmoglein 2 (DSG2) is a receptor for human adenovirus species B serotypes Ad3, Ad7, Ad11, and Ad14. Ad3 is considered to be a widely distributed human pathogen. Ad3 binding to DSG2 triggers the transient opening of epithelial junc …
We recently discovered that desmoglein 2 (DSG2) is a receptor for human adenovirus species B serotypes Ad3, Ad7, Ad11, and Ad14. A
Drosophila AD3 mutation of synaptotagmin impairs calcium-dependent self-oligomerization activity.
Fukuda M, Kabayama H, Mikoshiba K. Fukuda M, et al. FEBS Lett. 2000 Oct 6;482(3):269-72. doi: 10.1016/s0014-5793(00)02064-0. FEBS Lett. 2000. PMID: 11024474 Free article.
Genetic analysis of a Drosophila synaptotagmin (Syt) I mutant (AD3) has revealed that Tyr-334 within the C2B domain is essential for efficient Ca(2+)-dependent neurotransmitter release. ...This observation is consistent with the fact that the AD3 allele is homozygou …
Genetic analysis of a Drosophila synaptotagmin (Syt) I mutant (AD3) has revealed that Tyr-334 within the C2B domain is essential for …
Identification and expression analysis of a potential familial Alzheimer disease gene on chromosome 1 related to AD3.
Li J, Ma J, Potter H. Li J, et al. Proc Natl Acad Sci U S A. 1995 Dec 19;92(26):12180-4. doi: 10.1073/pnas.92.26.12180. Proc Natl Acad Sci U S A. 1995. PMID: 8618867 Free PMC article.
Recently, the gene likely responsible for this genetic linkage has been identified and termed AD3. Five mutations have been found in AD3 that segregate with the disease phenotype in seven AD families and are not present in unaffected individuals. ...The presence of …
Recently, the gene likely responsible for this genetic linkage has been identified and termed AD3. Five mutations have been found in …
581 results