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Activation of glutamate receptors promotes a calcium-dependent and transporter-mediated release of purines in cultured avian retinal cells: possible involvement of calcium/calmodulin-dependent protein kinase II.
Paes-de-Carvalho R, Dias BV, Martins RA, Pereira MR, Portugal CC, Lanfredi C. Paes-de-Carvalho R, et al. Neurochem Int. 2005 May;46(6):441-51. doi: 10.1016/j.neuint.2004.12.005. Neurochem Int. 2005. PMID: 15769546
Here we show that activation of NMDA or alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate (AMPA)/kainate glutamate ionotropic receptors promotes a two- to three-fold increase in the release of purines from these cultures. ...The regulation of adenosine release by glu
Here we show that activation of NMDA or alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate (AMPA)/kainate glutamate ionotropic rece …
Facilitatory effect of glutamate exocytosis from rat cerebrocortical nerve terminals by alpha-tocopherol, a major vitamin E component.
Yang TT, Wang SJ. Yang TT, et al. Neurochem Int. 2008 May;52(6):979-89. doi: 10.1016/j.neuint.2007.10.009. Epub 2007 Oct 18. Neurochem Int. 2008. PMID: 18037536
The effect of alpha-tocopherol, the major vitamin E component, on the release of endogenous glutamate has been investigated using rat cerebrocortical nerve terminals. ...In addition, alpha-tocopherol modulation of glutamate release appeared to involve a protein kina …
The effect of alpha-tocopherol, the major vitamin E component, on the release of endogenous glutamate has been investigated using rat …
Calcium-myristoyl switch, subcellular localization, and calcium-dependent translocation of the neuronal calcium sensor protein VILIP-3, and comparison with VILIP-1 in hippocampal neurons.
Spilker C, Braunewell KH. Spilker C, et al. Mol Cell Neurosci. 2003 Nov;24(3):766-78. doi: 10.1016/s1044-7431(03)00242-2. Mol Cell Neurosci. 2003. PMID: 14664824
Little is known about differences in the calcium-myristoyl switch in living cells leading to localization of VILIPs to distinct subcellular compartments. Therefore, we studied the calcium-dependent localization of green fluorescent protein (GFP)-tagged VILIP-3 in living ce …
Little is known about differences in the calcium-myristoyl switch in living cells leading to localization of VILIPs to distinct subce …
Substrate phosphorylation in the protein kinase Cgamma knockout mouse.
Ramakers GM, Gerendasy DD, de Graan PN. Ramakers GM, et al. J Biol Chem. 1999 Jan 22;274(4):1873-4. doi: 10.1074/jbc.274.4.1873. J Biol Chem. 1999. PMID: 9890937 Free article.
The phosphorylation state of three identified neural-specific protein kinase C substrates (RC3, GAP-43/B-50, and MARCKS) was monitored in hippocampal slices of mice lacking the gamma-subtype of protein kinase C and wild-type controls by quantitative immunoprecipitation following …
The phosphorylation state of three identified neural-specific protein kinase C substrates (RC3, GAP-43/B-50, and MARCKS) was monitored in hi …
(-)-Epigallocatechin gallate, the most active polyphenolic catechin in green tea, presynaptically facilitates Ca2+-dependent glutamate release via activation of protein kinase C in rat cerebral cortex.
Chou CW, Huang WJ, Tien LT, Wang SJ. Chou CW, et al. Synapse. 2007 Nov;61(11):889-902. doi: 10.1002/syn.20444. Synapse. 2007. PMID: 17663453
Results showed that the release of glutamate evoked by 4-aminopyridine (4AP) was facilitated by EGCG in a concentration-dependent manner, and this effect resulted from an enhancement of vesicular exocytosis and not from an increase in Ca2+-independent efflux via glutama
Results showed that the release of glutamate evoked by 4-aminopyridine (4AP) was facilitated by EGCG in a concentration-dependent man …
ATP:citrate lyase of Rhodotorula gracilis: purification and properties.
Shashi K, Bachhawat AK, Joseph R. Shashi K, et al. Biochim Biophys Acta. 1990 Jan 29;1033(1):23-30. doi: 10.1016/0304-4165(90)90189-4. Biochim Biophys Acta. 1990. PMID: 2302411
The enzyme was stimulated by NH+4 ions and inhibited by palmitoyl, lauroyl, oleoyl, myristoyl and stearoyl-CoA esters, glutamate and glucose 6-phosphate but not by acetyl-CoA or shorter chain fatty acyl-CoA esters. ...
The enzyme was stimulated by NH+4 ions and inhibited by palmitoyl, lauroyl, oleoyl, myristoyl and stearoyl-CoA esters, glutamate
Phosphorylation of synapsin I and MARCKS in nerve terminals is mediated by Ca2+ entry via an Aga-GI sensitive Ca2+ channel which is coupled to glutamate exocytosis.
Coffey ET, Sihra TS, Nicholls DG, Pocock JM. Coffey ET, et al. FEBS Lett. 1994 Oct 24;353(3):264-8. doi: 10.1016/0014-5793(94)01061-7. FEBS Lett. 1994. PMID: 7957871 Free article.
The novel spider toxin Aga-GI completely blocks KCl-evoked glutamate exocytosis but only partially inhibits KCl-evoked cytoplasmic Ca2+ elevations, thus revealing at least two pathways for KCl-induced Ca2+ entry. ...
The novel spider toxin Aga-GI completely blocks KCl-evoked glutamate exocytosis but only partially inhibits KCl-evoked cytoplasmic Ca …
Protein kinase C and the regulation of glutamate exocytosis from cerebrocortical synaptosomes.
Coffey ET, Sihra TS, Nicholls DG. Coffey ET, et al. J Biol Chem. 1993 Oct 5;268(28):21060-5. J Biol Chem. 1993. PMID: 8104937 Free article.
Although the PKC inhibitor Ro 31-8220 prevents both the depolarization-evoked and phorbol dibutyrate (PDBu)-evoked phosphorylation of the major presynaptic PKC substrate, myristoylated alanine-rich C kinase substrate, it is without effect on KCl-evoked Ca(2+)-dependent …
Although the PKC inhibitor Ro 31-8220 prevents both the depolarization-evoked and phorbol dibutyrate (PDBu)-evoked phosphorylation of the ma …