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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1957 2
1969 1
1971 1
1974 4
1975 4
1976 11
1977 5
1978 4
1979 4
1980 6
1981 9
1982 3
1983 9
1984 9
1985 13
1986 13
1987 9
1988 10
1989 8
1990 13
1991 11
1992 16
1993 11
1994 10
1995 12
1996 17
1997 9
1998 20
1999 20
2000 16
2001 8
2002 19
2003 23
2004 20
2005 22
2006 23
2007 11
2008 19
2009 13
2010 22
2011 18
2012 19
2013 18
2014 28
2015 24
2016 16
2017 20
2018 30
2019 17
2020 30
2021 25
2022 14
2023 7
2024 4

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677 results

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Quoted phrase not found in phrase index: "Abnormal nephron morphology"
Page 1
Pathophysiology of proteinuria.
D'Amico G, Bazzi C. D'Amico G, et al. Kidney Int. 2003 Mar;63(3):809-25. doi: 10.1046/j.1523-1755.2003.00840.x. Kidney Int. 2003. PMID: 12631062 Free article. Review.
Proteinuria is consequence of two mechanisms: the abnormal transglomerular passage of proteins due to increased permeability of glomerular capillary wall and their subsequent impaired reabsorption by the epithelial cells of the proximal tubuli. ...Recent clinical studies s …
Proteinuria is consequence of two mechanisms: the abnormal transglomerular passage of proteins due to increased permeability of glome …
Immunoglobulin A nephropathy in paediatrics: An up-to-date.
Paranhos RM, De Souza Figueiredo GA, De Abreu GR, Ferreira GC, Fonseca GG, Simões E Silva AC. Paranhos RM, et al. Nephrology (Carlton). 2022 Apr;27(4):307-317. doi: 10.1111/nep.13987. Epub 2021 Oct 29. Nephrology (Carlton). 2022. PMID: 34676611 Review.
It has been considered an autoimmune disease that can lead to the production of autoantibodies against abnormal IgA1 and formation of immune complexes. These autoantibodies and immune complexes deposit in the glomeruli, resulting in renal injury. ...The diagnosis is define …
It has been considered an autoimmune disease that can lead to the production of autoantibodies against abnormal IgA1 and formation of …
Clinical and pathological investigation of oligomeganephronia.
Kitakado H, Horinouchi T, Masuda C, Kondo A, Nagai S, Aoto Y, Sakakibara N, Ninchoji T, Yoshikawa N, Nozu K. Kitakado H, et al. Pediatr Nephrol. 2023 Mar;38(3):757-762. doi: 10.1007/s00467-022-05687-y. Epub 2022 Jul 21. Pediatr Nephrol. 2023. PMID: 35861872
BACKGROUND: Oligomeganephronia (OMN) is a rare congenital anomaly involving the kidney and urinary tract, characterized by decreased number and compensatory hypertrophy of the nephron. It is caused by abnormal kidney development during the embryonic period, e …
BACKGROUND: Oligomeganephronia (OMN) is a rare congenital anomaly involving the kidney and urinary tract, characterized by decreased …
Renal tubular dysgenesis.
Gubler MC. Gubler MC. Pediatr Nephrol. 2014 Jan;29(1):51-9. doi: 10.1007/s00467-013-2480-1. Epub 2013 May 1. Pediatr Nephrol. 2014. PMID: 23636579 Review.
Mutations result in either the absence of production or lack of efficacy of angiotensin II. Secondary RTD has been observed in various situations, particularly in the donor twin of severe twin-to-twin transfusion syndrome, in foetuses affected with congenital haemoc …
Mutations result in either the absence of production or lack of efficacy of angiotensin II. Secondary RTD has been observed in variou …
C3 glomerulopathy and current dilemmas.
Ito N, Ohashi R, Nagata M. Ito N, et al. Clin Exp Nephrol. 2017 Aug;21(4):541-551. doi: 10.1007/s10157-016-1358-5. Epub 2016 Nov 23. Clin Exp Nephrol. 2017. PMID: 27878657 Free PMC article. Review.
Recent molecular and genetic advances provide information to characterize C3G. Some C3G cases are found with genetic abnormalities in complement regulatory factors, but majority of cases seem to be associated with acquired factors that dysregulate the alternative complemen …
Recent molecular and genetic advances provide information to characterize C3G. Some C3G cases are found with genetic abnormalities in …
Familial hematuria: A review.
Plevová P, Gut J, Janda J. Plevová P, et al. Medicina (Kaunas). 2017;53(1):1-10. doi: 10.1016/j.medici.2017.01.002. Epub 2017 Jan 31. Medicina (Kaunas). 2017. PMID: 28236514 Free article. Review.
In X-linked Alport syndrome, a clear genotype-phenotype correlation is typically observed in men. Deleterious COL4A5 mutations are associated with a more severe renal phenotype and more frequent high-frequency sensorineural hearing loss and ocular abnormalities. ...
In X-linked Alport syndrome, a clear genotype-phenotype correlation is typically observed in men. Deleterious COL4A5 mutations are as …
New insights into the role of HNF-1β in kidney (patho)physiology.
Ferrè S, Igarashi P. Ferrè S, et al. Pediatr Nephrol. 2019 Aug;34(8):1325-1335. doi: 10.1007/s00467-018-3990-7. Epub 2018 Jul 1. Pediatr Nephrol. 2019. PMID: 29961928 Free PMC article. Review.
In the embryonic kidney, HNF-1beta is required for ureteric bud branching, initiation of nephrogenesis, and nephron segmentation. Ablation of mouse Hnf1b in nephron progenitors causes defective tubulogenesis, whereas later inactivation in elongating tubules leads to …
In the embryonic kidney, HNF-1beta is required for ureteric bud branching, initiation of nephrogenesis, and nephron segmentation. Abl …
Immunopathogenesis of idiopathic nephrotic syndrome with relapse.
Sahali D, Sendeyo K, Mangier M, Audard V, Zhang SY, Lang P, Ollero M, Pawlak A. Sahali D, et al. Semin Immunopathol. 2014 Jul;36(4):421-9. doi: 10.1007/s00281-013-0415-3. Epub 2014 Jan 9. Semin Immunopathol. 2014. PMID: 24402710 Free PMC article. Review.
Although the mechanisms underlying the pathophysiology of proteinuria remain unclear, clinical and experimental observations suggest that lymphocyte and podocyte disturbances are two sides of the disease. The current hypothesis suggests that immune cells release a putative …
Although the mechanisms underlying the pathophysiology of proteinuria remain unclear, clinical and experimental observations suggest …
Organoid-on-a-chip model of human ARPKD reveals mechanosensing pathomechanisms for drug discovery.
Hiratsuka K, Miyoshi T, Kroll KT, Gupta NR, Valerius MT, Ferrante T, Yamashita M, Lewis JA, Morizane R. Hiratsuka K, et al. Sci Adv. 2022 Sep 23;8(38):eabq0866. doi: 10.1126/sciadv.abq0866. Epub 2022 Sep 21. Sci Adv. 2022. PMID: 36129975 Free PMC article.
PKHD1-mutant organoids-on-a-chip are subjected to flow that induces clinically relevant phenotypes of distal nephron dilatation. Transcriptomics discover 229 signal pathways that are not identified by static models. ...Food and Drug Administration-approved and one investig …
PKHD1-mutant organoids-on-a-chip are subjected to flow that induces clinically relevant phenotypes of distal nephron dilatation. Tran …
Managing Complications Following Nephron-Sparing Procedures for Renal Masses.
Gonzalez-Aguirre AJ, Durack JC. Gonzalez-Aguirre AJ, et al. Tech Vasc Interv Radiol. 2016 Sep;19(3):194-202. doi: 10.1053/j.tvir.2016.06.004. Epub 2016 Jun 3. Tech Vasc Interv Radiol. 2016. PMID: 27641453 Review.
Small renal malignancies are commonly treated with nephron-sparing procedures including partial nephrectomy and percutaneous ablation. ...Here, we review vascular and nonvascular complications associated with nephron-sparing renal mass treatments and discuss options …
Small renal malignancies are commonly treated with nephron-sparing procedures including partial nephrectomy and percutaneous ablation …
677 results