Gao Y - Search Results

1

The proteasome activator REGγ promotes diabetic endothelial impairment by inhibiting HMGA2-GLUT1 pathway.

Xie Y, Gao R, Gao Y, Dong Z, Moses RE, Li X, Ge J. Xie Y, et al. Among authors: gao y, gao r. Transl Res. 2022 Aug;246:33-48. doi: 10.1016/j.trsl.2022.03.007. Epub 2022 Mar 31. Transl Res. 2022. PMID: 35367424

2

The proteasome activator REGγ accelerates cardiac hypertrophy by declining PP2Acα-SOD2 pathway.

Xie Y, Gao Y, Gao R, Yang W, Dong Z, Moses RE, Sun A, Li X, Ge J. Xie Y, et al. Among authors: gao y, gao r. Cell Death Differ. 2020 Oct;27(10):2952-2972. doi: 10.1038/s41418-020-0554-8. Epub 2020 May 18. Cell Death Differ. 2020. PMID: 32424140 Free PMC article.

3

GSDMD-Mediated Cardiomyocyte Pyroptosis Promotes Myocardial I/R Injury.

Shi H, Gao Y, Dong Z, Yang J, Gao R, Li X, Zhang S, Ma L, Sun X, Wang Z, Zhang F, Hu K, Sun A, Ge J. Shi H, et al. Among authors: gao y, gao r. Circ Res. 2021 Jul 23;129(3):383-396. doi: 10.1161/CIRCRESAHA.120.318629. Epub 2021 May 21. Circ Res. 2021. PMID: 34015941 Free PMC article.

4

The selective STING inhibitor H-151 preserves myocardial function and ameliorates cardiac fibrosis in murine myocardial infarction.

Hu S, Gao Y, Gao R, Wang Y, Qu Y, Yang J, Wei X, Zhang F, Ge J. Hu S, et al. Among authors: gao y, gao r. Int Immunopharmacol. 2022 Jun;107:108658. doi: 10.1016/j.intimp.2022.108658. Epub 2022 Mar 9. Int Immunopharmacol. 2022. PMID: 35278833

5

Current knowledge of pyroptosis in heart diseases.

Gao Y, Shi H, Dong Z, Zhang F, Sun A, Ge J. Gao Y, et al. J Mol Cell Cardiol. 2022 Oct;171:81-89. doi: 10.1016/j.yjmcc.2022.07.005. Epub 2022 Jul 20. J Mol Cell Cardiol. 2022. PMID: 35868567 Review.

6

Neutrophil-derived advanced glycation end products-Nε-(carboxymethyl) lysine promotes RIP3-mediated myocardial necroptosis via RAGE and exacerbates myocardial ischemia/reperfusion injury.

Yang J, Zhang F, Shi H, Gao Y, Dong Z, Ma L, Sun X, Li X, Chang S, Wang Z, Qu Y, Li H, Hu K, Sun A, Ge J. Yang J, et al. Among authors: gao y. FASEB J. 2019 Dec;33(12):14410-14422. doi: 10.1096/fj.201900115RR. Epub 2019 Oct 30. FASEB J. 2019. PMID: 31665609

In addition, glyoxalase-1 overexpression could effectively attenuate MI/RI by reducing CML formation, providing a potential therapeutic target for MI/RI.-Yang, J., Zhang, F., Shi, H., Gao, Y., Dong, Z., Ma, L., Sun, X., Li, X., Chang, S., Wang, Z., Qu, Y., Li …

In addition, glyoxalase-1 overexpression could effectively attenuate MI/RI by reducing CML formation, providing a potential therapeutic targ …

7

The selective NLRP3-inflammasome inhibitor MCC950 reduces myocardial fibrosis and improves cardiac remodeling in a mouse model of myocardial infarction.

Gao R, Shi H, Chang S, Gao Y, Li X, Lv C, Yang H, Xiang H, Yang J, Xu L, Tang Y. Gao R, et al. Among authors: gao y. Int Immunopharmacol. 2019 Sep;74:105575. doi: 10.1016/j.intimp.2019.04.022. Epub 2019 Jul 9. Int Immunopharmacol. 2019. PMID: 31299609

8

Qu Y, Yang J, Zhang F, Li C, Dai Y, Yang H, Gao Y, Pan Y, Yao K, Huang D, Lu H, Ma J, Qian J, Ge J. Qu Y, et al. Among authors: gao y. J Interv Cardiol. 2019 Jul 11;2019:6762089. doi: 10.1155/2019/6762089. eCollection 2019. J Interv Cardiol. 2019. PMID: 31772542 Free PMC article.

9

Optimal strategy of primary percutaneous coronary intervention for acute myocardial infarction due to unprotected left main coronary artery occlusion (OPTIMAL): study protocol for a randomised controlled trial.

Gao Y, Zhang F, Li C, Dai Y, Yang J, Qu Y, Qian J, Ge J; OPTIMAL trial investigators. Gao Y, et al. Trials. 2019 Mar 8;20(1):162. doi: 10.1186/s13063-019-3211-0. Trials. 2019. PMID: 30850023 Free PMC article.

10

Relationship between body mass index and outcomes of coronary artery disease in Asian population: Insight from the FOCUS registry.

Qu Y, Yang J, Zhang F, Li C, Dai Y, Yang H, Gao Y, Pan Y, Yao K, Huang D, Lu H, Ma J, Qian J, Ge J. Qu Y, et al. Among authors: gao y. Int J Cardiol. 2020 Feb 1;300:262-267. doi: 10.1016/j.ijcard.2019.10.025. Epub 2019 Oct 19. Int J Cardiol. 2020. PMID: 31685245 Clinical Trial.

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